p53 and tumor suppression

New England Journal of Medicine

Volumn 356, Issue 1, 2007, Pages 79-81

  Van Dyke, Terry ^a  

^a UNIVERSITY OF NORTH CAROLINA SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

4 HYDROXYTAMOXIFEN; ESTROGEN RECEPTOR; PROTEIN P53;

ARTICLE; CANCER GROWTH; CANCER INHIBITION; CARCINOGENESIS; CARCINOGENICITY; DNA DAMAGE; DNA RESPONSIVE ELEMENT; DRUG MECHANISM; HUMAN; IONIZING RADIATION; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN FUNCTION;

EID: 33845972685     PISSN: 00284793     EISSN: 15334406     Source Type: Journal    
DOI: 10.1056/NEJMcibr066301     Document Type: Article

References (4)

1. Christophorou MA, Ringshausen I, Finch AJ, Swigart LB, Evan GI. The pathological response to DNA damage does not contribute to p53-mediated tumour suppression. Nature 2006;443:214-7.
2. Efeyan A, Garcia-Cao I, Herranz D, Velasco-Miguel S, Serrano M. Tumour biology: policing of oncogene activity by p53. Nature 2006;443:159.
3. Christophorou MA, Martin-Zanca D, Soucek L, et al. Temporal dissection of p53 function in vitro and in vivo. Nat Genet 2005;37:718-26.
4. Garcia-Cao I, Garcia-Cao M, Martin-Caballero J, et al. "Super p53" mice exhibit enhanced DNA damage response, are tumor resistant and age normally. EMBO J 2002;21:6225-35.