Reply to commentary: Hepcidin mimetics from microorganisms? A possible explanation for the effect of Helicobacter pylori on iron homeostasis

Blood Cells, Molecules, and Diseases

Volumn 38, Issue 1, 2007, Pages 56-

  Hershko, Chaim ^a  

^a SHAARE ZEDEK MEDICAL CENTER   (Israel)

Author keywords

[No Author keywords available]

Indexed keywords

ASCORBIC ACID; FERROUS GLYCINE SULFATE; HEPCIDIN; HYDROCHLORIC ACID; IRON;

DISEASE ASSOCIATION; DRUG EFFICACY; DUODENUM MUCOSA; HELICOBACTER INFECTION; HELICOBACTER PYLORI; HUMAN; INFLAMMATION; IRON ABSORPTION; IRON BALANCE; IRON DEFICIENCY ANEMIA; IRON KINETICS; LETTER; MACROPHAGE; NONHUMAN; PRIORITY JOURNAL; STOMACH ACID SECRETION;

HELICOBACTER PYLORI;

EID: 33845682566     PISSN: 10799796     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.bcmd.2006.10.002     Document Type: Letter

References (2)

1. Nielsen P., Kongi R., Buggisch P., and Fischer R. Bioavailability of oral iron drugs as judged by a 59Fe-whole-body counting technique in patients with iron deficiency anaemia. Therapeutic efficacy of iron(II)-glycine sulfate. Arzneimittelforschung 55 (2005) 376-381
2. Annibale B., Capurso G., Lahner E., et al. Concomitant alterations in intragastric pH and ascorbic acid concentration in patients with Helicobacter pylori gastritis and associated iron deficiency anaemia. Gut 52 (2003) 496-501