Increased levels of myocardial IκB-α protein promote tolerance to endotoxin

American Journal of Physiology - Heart and Circulatory Physiology

Volumn 275, Issue 3 44-3, 1998, Pages

  Shames, Brian D ^a,b   Meldrum, Daniel R ^a   Selzman, Craig H ^a   Pulido, Edward J ^a   Cain, Brian S ^a   Banerjee, Anirban ^a   Harken, Alden H ^a   Meng, Xianzhong ^a  

^a UNIVERSITY OF COLORADO   (United States)

^b UNIVERSITY OF COLORADO   (United States)

Author keywords

Cardiac contractility; Glucocorticoids; Nuclear factor B; Rat; Tumor necrosis factor

Indexed keywords

BACTERIUM LIPOPOLYSACCHARIDE; DEXAMETHASONE; ENDOTOXIN; GLUCOCORTICOID; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; TUMOR NECROSIS FACTOR ALPHA;

ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; ENDOTOXEMIA; HEART FUNCTION; HEART MUSCLE CONTRACTILITY; IMMUNE RESPONSE; ISOLATED HEART; MALE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN PHOSPHORYLATION; RAT; SEPTIC SHOCK;

EID: 33750860428     PISSN: 03636135     EISSN: None     Source Type: Journal    
DOI: 10.1152/ajpheart.1998.275.3.h1084     Document Type: Article

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