The Barker hypothesis: Implications for future directions in toxicology research

Current Opinion in Endocrinology and Diabetes

Volumn 13, Issue 6, 2006, Pages 536-540

  Rogers, John M ^a,b  

^a U S ENVIRONMENTAL PROTECTION AGENCY   (United States)

^b Reproductive Toxicology Facility   (United States)

Author keywords

Barker hypothesis; Developmental toxicology; Fetal programming; Metabolic syndrome

Indexed keywords

ANTIOXIDANT; DEXAMETHASONE; DIETHYLSTILBESTROL; ESSENTIAL FATTY ACID; ESTRADIOL; GLUCOCORTICOID; VINCLOZOLIN;

BIRTH WEIGHT; CARBOHYDRATE DIET; DELAYED PUBERTY; DIET SUPPLEMENTATION; EPIGENETICS; FETUS DEVELOPMENT; GERM CELL; GLUCOSE INTOLERANCE; HUMAN; HYPERLEPTINEMIA; HYPERTENSION; HYPOTHESIS; LIPID DIET; LOW BIRTH WEIGHT; METABOLIC SYNDROME X; NONHUMAN; NUTRITION; PERINATAL PERIOD; PRENATAL EXPOSURE; PROGENY; PROTEIN RESTRICTION; REVIEW; TOXICOLOGY;

EID: 33750632603     PISSN: 10683097     EISSN: None     Source Type: Journal    
DOI: 10.1097/MED.0b013e328010d23b     Document Type: Review

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