Cardiac S100A1 protein levels determine contractile performance and propensity toward heart failure after myocardial infarction

Circulation

Volumn 114, Issue 12, 2006, Pages 1258-1268

  Most, Patrick ^a,b   Seifert, Hanna ^b   Gao, Erhe ^a   Funakoshi, Hajime ^a   Völkers, Mirko ^b   Heierhorst, Jörg ^c   Remppis, Andrew ^b   Pleger, Sven T ^a,b   DeGeorge Jr , Brent R ^a   Eckhart, Andrea D ^a   Feldman, Arthur M ^a   Koch, Walter J ^a  

^a THOMAS JEFFERSON UNIVERSITY   (United States)

^b UNIVERSITY OF HEIDELBERG   (Germany)

^c UNIVERSITY OF MELBOURNE   (Australia)

Author keywords

Calcium; Contractility; Heart failure; Myocardial infarction

Indexed keywords

BETA ADRENERGIC RECEPTOR; CALCIUM; GUANINE NUCLEOTIDE BINDING PROTEIN; PROTEIN KINASE C; PROTEIN S 100; PROTEIN S 100 A1; STIMULATORY GUANINE NUCLEOTIDE BINDING PROTEIN; UNCLASSIFIED DRUG;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CALCIUM CELL LEVEL; CONTROLLED STUDY; CORONARY ARTERY LIGATION; HEART FAILURE; HEART FUNCTION; HEART HYPERTROPHY; HEART INFARCTION; HEART INFARCTION SIZE; HEART MUSCLE CELL; HEART MUSCLE CONTRACTILE FORCE; HEART VENTRICLE PERFORMANCE; HEART VENTRICLE REMODELING; KNOCKOUT MOUSE; LEFT ANTERIOR DESCENDING CORONARY ARTERY; MOUSE; NONHUMAN; PATHOPHYSIOLOGY; PRIORITY JOURNAL; PROTEIN BLOOD LEVEL; PROTEIN EXPRESSION; SARCOPLASMIC RETICULUM; SPECIES COMPARISON; SURVIVAL TIME; TRANSGENIC MOUSE; WILD TYPE;

ANIMALS; APOPTOSIS; CALCIUM-TRANSPORTING ATPASES; CARDIAC OUTPUT, LOW; CYCLIC AMP; DISEASE PROGRESSION; DOWN-REGULATION; GTP-BINDING PROTEIN ALPHA SUBUNITS, GS; MICE; MICE, KNOCKOUT; MICE, TRANSGENIC; MYOCARDIAL CONTRACTION; MYOCARDIAL INFARCTION; MYOCARDIUM; RECEPTORS, ADRENERGIC, BETA; RNA, MESSENGER; S100 PROTEINS; SARCOPLASMIC RETICULUM CALCIUM-TRANSPORTING ATPASES; VENTRICULAR REMODELING;

EID: 33748754119     PISSN: 00097322     EISSN: None     Source Type: Journal    
DOI: 10.1161/CIRCULATIONAHA.106.622415     Document Type: Article

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