Clock genes as a link between addiction and obesity [1]

European Journal of Human Genetics

Volumn 14, Issue 1, 2006, Pages 5-

  Manev, Hari ^a   Uz, Tolga ^a  

^a UNIVERSITY OF ILLINOIS AT CHICAGO   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

DOPAMINE; FATTY ACID; PROTEIN BMAL1; TRANSCRIPTION FACTOR CLOCK;

ADIPOSE TISSUE CELL; COCAINE DEPENDENCE; DOPAMINERGIC SYSTEM; DRUG DEPENDENCE; E BOX ELEMENT; EATING; FATTY ACID METABOLISM; GENE EXPRESSION; GENE FUNCTION; HUMAN; LETTER; LIPOGENESIS; METABOLIC SYNDROME X; MOUSE MUTANT; NONHUMAN; OBESITY; PATHOGENESIS; PHENOTYPE; PRIORITY JOURNAL; PROMOTER REGION; REWARD;

ANIMALS; BASIC HELIX-LOOP-HELIX TRANSCRIPTION FACTORS; CIRCADIAN RHYTHM; COCAINE; MICE; MICE, MUTANT STRAINS; OBESITY; SUBSTANCE-RELATED DISORDERS; TRANS-ACTIVATORS;

EID: 33748066767     PISSN: 10184813     EISSN: 14765438     Source Type: Journal    
DOI: 10.1186/1747-5333-1-5     Document Type: Letter

References (5)

1. Yuferov V, Butelman ER, Kreek MJ: Biological clock: Biological clocks may modulate drug addiction. Eur J Hum Genet 2005; 13: 1101-1103.
2. McClung CA, Sidiropoulou K, Vitaterna M et al: Regulation of dopaminergic transmission and cocaine reward by the Clock gene. Proc Natl Acad Sci USA 2005; 102: 9377-9381.
3. Turek FW, Joshu C, Kohsaka A et al: Obesity and metabolic syndrome in circadian Clock mutant mice. Science 2005; 308: 1043-1045.
4. Shimba S, Ishii N, Ohta Y et al: Brain and muscle Arnt-like protein-1 (BMAL1), a component of the molecular clock, regulates adipogenesis. Proc Natl Acad Sci USA 2005; 102: 12071-12076.
5. Kurtuncu M, Battista N, Uz T et al: 5-Lipoxygenase gene disruption in mice alters their behavior, cocaine responsiveness, and the brain endocannabinoid system. Program No. 800.6, 2005 Abstract Viewer/ Itinerary Planner. Washington, DC: Society for Neuroscience, Online.