GSK-3β as a driving force in ovarian cancer

Cell Research

Volumn 16, Issue 7, 2006, Pages 609-

  Li, Dangsheng ^a  

^a Shanghai Jiao Tong University Affiliated Sixth People s Hospital   (China)

Author keywords

[No Author keywords available]

Indexed keywords

GLYCOGEN SYNTHASE KINASE 3; GLYCOGEN SYNTHASE KINASE 3 BETA;

ANIMAL; CELL PROLIFERATION; ENZYMOLOGY; FEMALE; HUMAN; METABOLISM; MOUSE; NOTE; OVARY TUMOR; TUMOR CELL LINE;

ANIMALS; CELL LINE, TUMOR; CELL PROLIFERATION; FEMALE; GLYCOGEN SYNTHASE KINASE 3; HUMANS; MICE; OVARIAN NEOPLASMS;

EID: 33746218416     PISSN: 10010602     EISSN: 17487838     Source Type: Journal    
DOI: 10.1038/sj.cr.7310082     Document Type: Article

References (3)

1. Cao Q, Lu X, Feng YJ. Glycogen synthase kinase-3β positively regulates the proliferation of human ovarian cancer cells. Cell Res 2006; 16:671-677.
2. Ougolkov AV, Fernandez-Zapico ME, Savoy DN, Urrutia RA, Billadeau DD. Glycogen synthase kinase-3β participates in nuclear factor κB-mediated gene transcription and cell survival in pancreatic cancer cells. Cancer Res 2005; 65:2076-2081.
3. Shakoori A, Ougolkov AV, Yu ZW, et al. Deregulated GSK3β activity in colorectal cancer: its association with tumor cell survival and proliferation. Biochem Biophys Res Commun 2005; 334:1365-1373.