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Volumn 25, Issue 29, 2006, Pages 4016-4026
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The mutation in the ATP-binding region of JAK1, identified in human uterine leiomyosarcomas, results in defective interferon-γ inducibility of TAP1 and LMP2
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Author keywords
IFN ; JAK1; Leiomyosarcoma; LMP2; Uterus
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Indexed keywords
ADENOSINE TRIPHOSPHATE;
GAMMA INTERFERON;
JANUS KINASE 1;
PROTEIN;
PROTEIN LMP2;
TRANSPORTER ASSOCIATED WITH ANTIGEN PROCESSING 1;
UNCLASSIFIED DRUG;
ANTIGEN PRESENTATION;
ARTICLE;
CANCER CELL CULTURE;
CONTROLLED STUDY;
CYTOKINE RELEASE;
GENE MUTATION;
HUMAN;
HUMAN CELL;
LEIOMYOSARCOMA;
NUCLEOTIDE SEQUENCE;
PRIORITY JOURNAL;
PROTEIN EXPRESSION;
PROTEIN INDUCTION;
SIGNAL TRANSDUCTION;
UPREGULATION;
UTERUS SARCOMA;
ADENOSINE TRIPHOSPHATE;
AMINO ACID SUBSTITUTION;
ANIMALS;
ANTIGEN PRESENTATION;
ANTIGENS, NEOPLASM;
ATP-BINDING CASSETTE TRANSPORTERS;
CYSTEINE ENDOPEPTIDASES;
FEMALE;
GENE EXPRESSION REGULATION, NEOPLASTIC;
GENES, MHC CLASS I;
HELA CELLS;
HUMANS;
IMMUNOTHERAPY;
INTERFERON TYPE II;
JANUS KINASE 1;
LEIOMYOMA;
MICE;
MICE, KNOCKOUT;
POINT MUTATION;
PROTEIN BINDING;
PROTEIN STRUCTURE, TERTIARY;
PROTEIN-TYROSINE KINASES;
SARCOMA;
SIGNAL TRANSDUCTION;
TUMOR ESCAPE;
UTERINE NEOPLASMS;
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EID: 33745793005
PISSN: 09509232
EISSN: 14765594
Source Type: Journal
DOI: 10.1038/sj.onc.1209434 Document Type: Article |
Times cited : (40)
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References (34)
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