Transient recovery of synaptic transmission is related to rapid energy depletion during hypoxia

Neuroscience Letters

Volumn 400, Issue 1-2, 2006, Pages 1-6

  Kim, Joo Han ^a   Park, Youn Kwan ^a   Kim, Jong Hyun ^a   Kwon, Taek Hyun ^a   Chung, Hung Seob ^a  

^a Korea University College of Medicine   (South Korea)

Author keywords

Hippocampus; Hypoglycemia; Hypoxia; In vitro; Synaptic transmission

Indexed keywords

CALCIUM ION; LACTIC ACID; PROTEIN KINASE C;

ANIMAL TISSUE; ARTICLE; BRAIN HYPOXIA; BRAIN TISSUE; CALCIUM SIGNALING; CONTROLLED STUDY; DEPOLARIZATION; ENERGY EXPENDITURE; ENERGY YIELD; EVOKED CORTICAL RESPONSE; GLUCOSE BLOOD LEVEL; GLYCOLYSIS; HIPPOCAMPUS; HYPOGLYCEMIA; MALE; NEUROPATHOLOGY; NONHUMAN; OXIDATIVE PHOSPHORYLATION; PRIORITY JOURNAL; RAT; SYNAPTIC TRANSMISSION; TISSUE SLICE;

ANIMALS; ANOXIA; CHELATING AGENTS; DANTROLENE; DOSE-RESPONSE RELATIONSHIP, DRUG; EGTAZIC ACID; ELECTRIC STIMULATION; ENZYME INHIBITORS; EXCITATORY POSTSYNAPTIC POTENTIALS; GLUCOSE; HIPPOCAMPUS; HYPOGLYCEMIA; MALE; PHORBOL ESTERS; POTASSIUM CHLORIDE; RATS; RATS, SPRAGUE-DAWLEY; SYNAPSES; SYNAPTIC TRANSMISSION; TIME FACTORS;

EID: 33646159952     PISSN: 03043940     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.neulet.2006.01.035     Document Type: Article

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