v-Src requires Ras signaling for the suppression of gap junctional intercellular communication

Oncogene

Volumn 25, Issue 16, 2006, Pages 2420-2424

  Ito, S ^a   Ito, Y ^a   Senga, T ^a   Hattori, S ^b   Matsuo, S ^a   Hamaguchi, M ^a  

^a NAGOYA UNIVERSITY GRADUATE SCHOOL OF MEDICINE   (Japan)

^b UNIVERSITY OF TOKYO   (Japan)

Author keywords

Cell cell communication; Connexin43; Gap junction; Ras; v Src

Indexed keywords

COMPLEMENTARY DNA; CONNEXIN 43; MANUMYCIN; PROTEIN FARNESYLTRANSFERASE INHIBITOR; RAS PROTEIN; TETRACYCLINE;

ANIMAL CELL; ARTICLE; CELL COMMUNICATION; CELL TRANSFORMATION; ENZYME PHOSPHORYLATION; GAP JUNCTION; MEMBRANE CHANNEL; NONHUMAN; ONCOGENE RAS; PRIORITY JOURNAL; RAT; SIGNAL TRANSDUCTION;

ANIMALS; CELL COMMUNICATION; CELL LINE; CELL TRANSFORMATION, NEOPLASTIC; CONNEXIN 43; GAP JUNCTIONS; ONCOGENE PROTEIN PP60(V-SRC); RAS PROTEINS; RATS; SIGNAL TRANSDUCTION;

ANIMALIA;

EID: 33646130857     PISSN: 09509232     EISSN: 14765594     Source Type: Journal    
DOI: 10.1038/sj.onc.1209263     Document Type: Article

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