The negative role of cyclin G in ATM-dependent p53 activation

Oncogene

Volumn 23, Issue 31, 2004, Pages 5405-5408

  Ohtsuka, Takao ^a   Jensen, Michael R ^b   Hyung, Gu Kim ^a   Kim, Kyung Tae ^a   Lee, Sam W ^a,c  

^a HARVARD MEDICAL SCHOOL   (United States)

^b FIRC INSTITUTE OF MOLECULAR ONCOLOGY   (Italy)

^c HARVARD INSTITUTES OF MEDICINE   (United States)

Author keywords

ATM; Cell cycle; Cyclin G; DNA damage; p53

Indexed keywords

ATM PROTEIN; CYCLIN G; CYCLINE; DNA; PROTEIN P53; SERINE; UNCLASSIFIED DRUG; CELL CYCLE PROTEIN; DNA BINDING PROTEIN; GREEN FLUORESCENT PROTEIN; PHOTOPROTEIN; PROTEIN SERINE THREONINE KINASE; TUMOR SUPPRESSOR PROTEIN;

ANTIGEN EXPRESSION; ARTICLE; CELL CYCLE; CELL NUCLEUS; CONTROLLED STUDY; DNA DAMAGE; FIBROBLAST; GENE MUTATION; GENE TRANSLOCATION; HUMAN; HUMAN CELL; MITOSIS INHIBITION; PRIORITY JOURNAL; PROTEIN PHOSPHORYLATION; REDUCTION; SKIN CELL; STEADY STATE; CHEMISTRY; FLOW CYTOMETRY; GENETIC TRANSFECTION; METABOLISM; MUTATION; PHOSPHORYLATION; PHYSIOLOGY; PLASMID;

ATAXIA; ATAXIA TELANGIECTASIA;

CELL CYCLE; CELL CYCLE PROTEINS; CYCLINS; DNA DAMAGE; DNA-BINDING PROTEINS; FLOW CYTOMETRY; GREEN FLUORESCENT PROTEINS; HUMANS; LUMINESCENT PROTEINS; MUTATION; PHOSPHORYLATION; PLASMIDS; PROTEIN-SERINE-THREONINE KINASES; SERINE; TRANSFECTION; TUMOR SUPPRESSOR PROTEIN P53; TUMOR SUPPRESSOR PROTEINS;

EID: 3142679562     PISSN: 09509232     EISSN: None     Source Type: Journal    
DOI: 10.1038/sj.onc.1207693     Document Type: Article

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