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Volumn 26, Issue 1, 2006, Pages 20-23

Toxic effects of hyperhomocysteinemia in chronic renal failure and in uremia: Cardiovascular and metabolic consequences

Author keywords

Chronic renal failure; Folate receptors; Homocysteine; Hypomethylation; Uremia; Uremic toxins

Indexed keywords

ISOASPARTIC ACID; MEMBRANE PROTEIN;

EID: 30444434397     PISSN: 02709295     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.semnephrol.2005.06.005     Document Type: Article
Times cited : (8)

References (12)
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  • 2
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    • A low, rather than a high, total plasma homocysteine is an indicator of poor outcome in hemodialysis patients
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  • 3
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    • Randomized trial of folic acid for prevention of cardiovascular events in end-stage renal disease
    • E.M. Wrone, J.M. Hornberger, J.L. Zehnder Randomized trial of folic acid for prevention of cardiovascular events in end-stage renal disease J Am Soc Nephrol 15 2004 420 426
    • (2004) J Am Soc Nephrol , vol.15 , pp. 420-426
    • Wrone, E.M.1    Hornberger, J.M.2    Zehnder, J.L.3
  • 4
    • 1342268311 scopus 로고    scopus 로고
    • Translational upregulation of folate receptors is mediated by homocysteine via RNA-heterogeneous nuclear ribonucleoprotein E1 interactions
    • A.C. Antony, Y. Tang, R.A. Khan Translational upregulation of folate receptors is mediated by homocysteine via RNA-heterogeneous nuclear ribonucleoprotein E1 interactions J Clin Invest 113 2004 285 301
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    • Antony, A.C.1    Tang, Y.2    Khan, R.A.3
  • 5
    • 10544252271 scopus 로고    scopus 로고
    • Hyperhomocysteinemia confers an independent increased risk of atherosclerosis in end-stage renal disease and is closely linked to plasma folate and pyridoxine concentrations
    • K. Robinson, A. Gupta, V. Dennis Hyperhomocysteinemia confers an independent increased risk of atherosclerosis in end-stage renal disease and is closely linked to plasma folate and pyridoxine concentrations Circulation 94 1996 2743 2748
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    • Robinson, K.1    Gupta, A.2    Dennis, V.3
  • 7
    • 0028919731 scopus 로고
    • Mechanism of erythrocyte accumulation of methylation inhibitor S-adenosylhomocysteine in uremia
    • A.F. Perna, D. Ingrosso, N.G. De Santo Mechanism of erythrocyte accumulation of methylation inhibitor S-adenosylhomocysteine in uremia Kidney Int 47 1995 247 253
    • (1995) Kidney Int , vol.47 , pp. 247-253
    • Perna, A.F.1    Ingrosso, D.2    De Santo, N.G.3
  • 8
    • 0038663165 scopus 로고    scopus 로고
    • Folate treatment and unbalanced methylation and changes of allelic expression induced by hyperhomocysteinaemia in patients with uremia
    • D. Ingrosso, A. Cimmino, A.F. Perna Folate treatment and unbalanced methylation and changes of allelic expression induced by hyperhomocysteinaemia in patients with uremia Lancet 361 2003 1693 1699
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  • 9
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    • D-aspartate content of erythrocyte membrane proteins is decreased in uremia: Implications for the repair of damaged proteins
    • A.F. Perna, A. D'Aniello, J.D. Lowenson D-aspartate content of erythrocyte membrane proteins is decreased in uremia Implications for the repair of damaged proteins J Am Soc Nephrol 8 1997 95 104
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  • 10
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    • Plasma protein aspartyl damage is increased in hemodialysis patients: Studies on causes and consequences
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.