The Fas receptor - 1,4,5-IP3 cascade: A potential target for treating heart failure and arrhythmias

Annals of the New York Academy of Sciences

Volumn 1015, Issue , 2004, Pages 338-350

  Binah, Ofer ^a   Shilkrut, Mark ^a   Yaniv, Gal ^a   Larisch, Sarit ^b  

^a TECHNION ISRAEL INSTITUTE OF TECHNOLOGY   (Israel)

^b RAMBAM MEDICAL CENTER   (Israel)

Author keywords

1,4,5 IP3; Apoptosis; Arrhythmias; Fas receptor; Heart failure; Hypoxia; Ventricular myocytes

Indexed keywords

CALCIUM; CASPASE INHIBITOR; FAS ANTIGEN; GENISTIN; PHOSPHOLIPASE C; PROTEIN TYROSINE KINASE;

ACTION POTENTIAL; ANIMAL TISSUE; APOPTOSIS; CALCIUM TRANSPORT; CELL MEMBRANE DEPOLARIZATION; CONFERENCE PAPER; CONTROLLED STUDY; DIASTOLIC BLOOD PRESSURE; HEART ARRHYTHMIA; HEART FAILURE; HEART MUSCLE CELL; HEART VENTRICLE; HYPOXIA; MEMBRANE STEADY POTENTIAL; NEWBORN; NONHUMAN; PROTEIN EXPRESSION; PROTEIN PHOSPHORYLATION; RAT;

ANIMALIA;

EID: 3042833684     PISSN: 00778923     EISSN: None     Source Type: Book Series    
DOI: 10.1196/annals.1302.030     Document Type: Conference Paper

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