Effect of bax deletion on ethanol sensitivity in the neonatal rat cerebellum

Journal of Neurobiology

Volumn 66, Issue 1, 2006, Pages 95-101

  Heaton, Marieta Barrow ^a,b   Paiva, Michael ^a,b   Madorsky, Irina ^a,b   Siler Marsiglio, Kendra ^a,b   Shaw, Gerry ^a,b  

^a UNIVERSITY OF FLORIDA COLLEGE OF MEDICINE   (United States)

^b Center for Alcohol Research   (United States)

Author keywords

bax; Cerebellum; Fetal alcohol syndrome; Knock out; Reactive oxygen species

Indexed keywords

ALCOHOL; INHIBITOR OF APOPTOSIS PROTEIN; PROTEIN BAX; PROTEIN BCL 2; REACTIVE OXYGEN METABOLITE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; BAX GENE; BRAIN DEVELOPMENT; CELL COUNT; CELL LOSS; CEREBELLUM; CONTROLLED STUDY; EXPOSURE; GENE; GENE DELETION; GENE TARGETING; GRANULE CELL; KNOCKOUT MOUSE; MOUSE; MULTIGENE FAMILY; NEUROPROTECTION; NEUROTOXICITY; NEWBORN; NEWBORN PERIOD; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; PURKINJE CELL; WILD TYPE;

ADMINISTRATION, INHALATION; ANALYSIS OF VARIANCE; ANIMALS; ANIMALS, GENETICALLY MODIFIED; ANIMALS, NEWBORN; BCL-2-ASSOCIATED X PROTEIN; BEHAVIOR, ANIMAL; CELL COUNT; CENTRAL NERVOUS SYSTEM DEPRESSANTS; CEREBELLUM; ETHANOL; IMAGING, THREE-DIMENSIONAL; MICE; NEURONS; REACTIVE OXYGEN SPECIES;

EID: 29244460669     PISSN: 00223034     EISSN: None     Source Type: Journal    
DOI: 10.1002/neu.20208     Document Type: Article

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