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Volumn 8, Issue 7, 2005, Pages 855-857
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Loss of mammalian Sprouty2 leads to enteric neuronal hyperplasia and esophageal achalasia
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Author keywords
[No Author keywords available]
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Indexed keywords
GLIAL CELL LINE DERIVED NEUROTROPHIC FACTOR;
MITOGEN ACTIVATED PROTEIN KINASE 3;
MONOCLONAL ANTIBODY;
PROTEIN;
PROTEIN KINASE B;
PROTEIN SPROUTY 2;
UNCLASSIFIED DRUG;
ANTIBODY;
GDNF PROTEIN, MOUSE;
MITOGEN ACTIVATED PROTEIN KINASE;
NERVE GROWTH FACTOR;
ONCOPROTEIN;
PROTEIN SERINE THREONINE KINASE;
SPRY2 PROTEIN, MOUSE;
ANIMAL EXPERIMENT;
ANIMAL TISSUE;
ARTICLE;
CELL SURVIVAL;
CONTROLLED STUDY;
ESOPHAGUS ACHALASIA;
INTESTINE PSEUDOOBSTRUCTION;
MAMMAL;
MOUSE;
NERVE CELL;
NERVE MALFORMATION;
NONHUMAN;
PERINATAL DEVELOPMENT;
PRIORITY JOURNAL;
ANIMAL;
ENZYME ACTIVATION;
GENE DELETION;
GENETICS;
HYPERPLASIA;
IMMUNOLOGY;
INTESTINE INNERVATION;
METABOLISM;
MOUSE MUTANT;
PATHOLOGY;
ANIMALS;
ANTIBODIES;
ENTERIC NERVOUS SYSTEM;
ENZYME ACTIVATION;
ESOPHAGEAL ACHALASIA;
EXTRACELLULAR SIGNAL-REGULATED MAP KINASES;
GENE DELETION;
GLIAL CELL LINE-DERIVED NEUROTROPHIC FACTOR;
HYPERPLASIA;
MICE;
MICE, KNOCKOUT;
NERVE GROWTH FACTORS;
NEURONS;
PROTEIN-SERINE-THREONINE KINASES;
PROTEINS;
PROTO-ONCOGENE PROTEINS;
PROTO-ONCOGENE PROTEINS C-AKT;
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EID: 24944448766
PISSN: 10976256
EISSN: None
Source Type: Journal
DOI: 10.1038/nn1485 Document Type: Article |
Times cited : (122)
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References (11)
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