Apoptotic cells quench reactive oxygen and nitrogen species and modulate TNF-α/TGF-β1 balance in activated macrophages: Involvement of phosphatidylserine-dependent and -independent pathways [1]

Cell Death and Differentiation

Volumn 12, Issue 8, 2005, Pages 1141-1144

  Serinkan, B F ^a   Gambelli, F ^a   Potapovich, Al ^a   Babu, H ^a   Di Giuseppe, M ^a   Ortiz, L A ^a   Fabisiak, J P ^a   Kagan, V E ^a  

^a UNIVERSITY OF PITTSBURGH   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

INDUCIBLE NITRIC OXIDE SYNTHASE; MITOGEN ACTIVATED PROTEIN KINASE; PHOSPHATIDYLSERINE; REACTIVE NITROGEN SPECIES; REACTIVE OXYGEN METABOLITE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE PHOSPHATE OXIDASE; TRANSCRIPTION FACTOR; TRANSFORMING GROWTH FACTOR BETA1; TUMOR NECROSIS FACTOR ALPHA;

APOPTOSIS; CELL TRANSPORT; CYTOKINE PRODUCTION; ENZYME ACTIVATION; HUMAN; IMMUNE RESPONSE; IMMUNOFLUORESCENCE; INFLAMMATION; LETTER; MACROPHAGE ACTIVATION; MOLECULAR MECHANICS; NONHUMAN; OXIDATION REDUCTION STATE; PRIORITY JOURNAL; REGULATORY MECHANISM; SIGNAL TRANSDUCTION;

ANIMALS; APOPTOSIS; HUMANS; JURKAT CELLS; MACROPHAGE ACTIVATION; MACROPHAGES; MICE; PHOSPHATIDYLSERINES; REACTIVE NITROGEN SPECIES; REACTIVE OXYGEN SPECIES; TRANSFORMING GROWTH FACTOR BETA; TUMOR NECROSIS FACTOR-ALPHA;

EID: 23044481743     PISSN: 13509047     EISSN: None     Source Type: Journal    
DOI: 10.1038/sj.cdd.4401619     Document Type: Letter

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