Lack of Fas/CD95 surface expression in highly proliferative leukemic cell lines correlates with loss of CtBP/BARS and redirection of the protein toward giant lysosomal structures

Cell Growth and Differentiation

Volumn 13, Issue 7, 2002, Pages 315-324

  Monleón, Inmaculada ^a   Iturralde, María ^a   Martínez Lorenzo, María José ^a   Monteagudo, Luis ^a   Lasierra, Pilar ^a   Larrad, Luis ^a   Piñeiro, Andrés ^a   Naval, Javier ^a   Alava, María Angeles ^a   Anel, Alberto ^a  

^a UNIVERSITY OF ZARAGOZA   (Spain)

Author keywords

[No Author keywords available]

Indexed keywords

ARACHIDONIC ACID; CELL PROTEIN; ENDOPHILIN I; FAS ANTIGEN; INTERLEUKIN 2 RECEPTOR; LYSOPHOSPHATIDIC ACID; PROTEIN BARS; PROTEIN CTBP; UNCLASSIFIED DRUG; ALCOHOL DEHYDROGENASE; C TERMINAL BINDING PROTEIN; C-TERMINAL BINDING PROTEIN; CARRIER PROTEIN; CD3 ANTIGEN; CTBP1 PROTEIN, RAT; DNA BINDING PROTEIN; HYDROLASE; LEUKOCYTE ANTIGEN; LYSOSOME ASSOCIATED MEMBRANE PROTEIN; MEMBRANE LIPID; MONENSIN; PHOSPHOPROTEIN; SERINE PROTEINASE INHIBITOR; SERPINA3 PROTEIN, BOS TAURUS; TRANSCRIPTION FACTOR;

ACYLATION; ANTIGEN EXPRESSION; ARACHIDONIC ACID METABOLISM; ARTICLE; CELL VACUOLE; CONTROLLED STUDY; HUMAN; HUMAN CELL; LEUKEMIA CELL LINE; LYSOSOME; PRIORITY JOURNAL; PROTEIN EXPRESSION; CELL DIVISION; CELL MEMBRANE; CELL TRANSFORMATION; CYTOPLASM; DRUG ANTAGONISM; ELECTRON MICROSCOPY; GENE EXPRESSION REGULATION; GENETICS; LEUKEMIA; METABOLISM; PATHOPHYSIOLOGY; PHYSIOLOGY; ULTRASTRUCTURE;

ANTIGENS, CD; ANTIGENS, CD3; ANTIGENS, CD95; ARACHIDONIC ACID; CARRIER PROTEINS; CELL DIVISION; CELL MEMBRANE; CELL TRANSFORMATION, NEOPLASTIC; CYTOPLASM; DNA-BINDING PROTEINS; GENE EXPRESSION REGULATION, LEUKEMIC; HUMAN; HYDROLASES; JURKAT CELLS; LEUKEMIA; LYSOSOMES; MEMBRANE LIPIDS; MICROSCOPY, ELECTRON; MONENSIN; PHOSPHOPROTEINS; SERPINS; SUPPORT, NON-U.S. GOV'T; ALCOHOL OXIDOREDUCTASES; HUMANS; LYSOSOME-ASSOCIATED MEMBRANE GLYCOPROTEINS; TRANSCRIPTION FACTORS;

EID: 18444401750     PISSN: 10449523     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

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