-
2
-
-
0025630642
-
Similar frequencies of renin gene restriction fragment length polymorphisms in hypertensive and normotensive subjects
-
Soubrier F, Jeunemaitre X, Rigat B, Houot AM, Cambien F, Corvol P. Similar frequencies of renin gene restriction fragment length polymorphisms in hypertensive and normotensive subjects. Hypertension 1990; 16: 712-7.
-
(1990)
Hypertension
, vol.16
, pp. 712-717
-
-
Soubrier, F.1
Jeunemaitre, X.2
Rigat, B.3
Houot, A.M.4
Cambien, F.5
Corvol, P.6
-
3
-
-
0026337814
-
Control of coronary vasomatar tone in ischaemic myocardium by local metabolism and neurohumoral mechanisms
-
Heusch G. Control of coronary vasomatar tone in ischaemic myocardium by local metabolism and neurohumoral mechanisms. Eur Heart J 1991; 12 (Suppl. F): 99-106.
-
(1991)
Eur Heart J
, vol.12
, Issue.SUPPL. F
, pp. 99-106
-
-
Heusch, G.1
-
4
-
-
0034651799
-
Alpha-adreneraic coronary vasoconstriction and myocardial ischemia in humans
-
Heusch G, Baumgart D, Camici P et al. Alpha-adreneraic coronary vasoconstriction and myocardial ischemia in humans. Circulation 2000; 101: 689-94.
-
(2000)
Circulation
, vol.101
, pp. 689-694
-
-
Heusch, G.1
Baumgart, D.2
Camici, P.3
-
5
-
-
0037438803
-
Emerging importance of alpha-adrenergic coronary vasoconstriction in acute coronary syndromes and its genetic background
-
Heusch G. Emerging importance of alpha-adrenergic coronary vasoconstriction in acute coronary syndromes and its genetic background. J Am Coll Cardiol 2003; 41: 195-6.
-
(2003)
J Am Coll Cardiol
, vol.41
, pp. 195-196
-
-
Heusch, G.1
-
6
-
-
0030482532
-
Angiotensin-converting enzyme inhibition restores flow-dependent and cold pressor test-induced dilations in coronary arteries of hypertensive patients
-
Antony I, Lerebours G, Nitenberg A. Angiotensin-converting enzyme inhibition restores flow-dependent and cold pressor test-induced dilations in coronary arteries of hypertensive patients. Circulation 1996; 94: 3115-22.
-
(1996)
Circulation
, vol.94
, pp. 3115-3122
-
-
Antony, I.1
Lerebours, G.2
Nitenberg, A.3
-
7
-
-
9444280103
-
Angiotensin-converting enzyme inhibition with quinapril improves endothelial vasomotor dysfunction in patients with coronary artery disease. The TREND (Trial on Reversing ENdothelial Dysfunction) Study
-
Mancini GB, Henry GC, Macaya C et al. Angiotensin-converting enzyme inhibition with quinapril improves endothelial vasomotor dysfunction in patients with coronary artery disease. The TREND (Trial on Reversing ENdothelial Dysfunction) Study. Circulation 1996; 94: 258-65.
-
(1996)
Circulation
, vol.94
, pp. 258-265
-
-
Mancini, G.B.1
Henry, G.C.2
Macaya, C.3
-
8
-
-
0033101753
-
Abnormal flow-mediated epicardial vasomotion in human coronary arteries is improved by angiotensin-converting enzyme inhibition: A potential role of bradykinin
-
Prasad A, Husain S, Quyyumi AA. Abnormal flow-mediated epicardial vasomotion in human coronary arteries is improved by angiotensin-converting enzyme inhibition: a potential role of bradykinin. J Am Coll Cardiol 1999; 33: 796-804.
-
(1999)
J Am Coll Cardiol
, vol.33
, pp. 796-804
-
-
Prasad, A.1
Husain, S.2
Quyyumi, A.A.3
-
9
-
-
0042330455
-
Efficacy of perindopril in reduction of cardiovascular events among patients with stable coronary artery disease: Randomised double-blind, placebo-controlled, multicenter trial (the EUROPA study)
-
EURopean trial On reduction of cardiac events with Perindopril in stable coronary Artery disease Investigators
-
Fox KM. EURopean trial On reduction of cardiac events with Perindopril in stable coronary Artery disease Investigators. Efficacy of perindopril in reduction of cardiovascular events among patients with stable coronary artery disease: randomised double-blind, placebo-controlled, multicenter trial (the EUROPA study). Lancet. 2003; 362: 782-8.
-
(2003)
Lancet
, vol.362
, pp. 782-788
-
-
Fox, K.M.1
-
10
-
-
0026675062
-
Deletion polymorphism in the gene for angiotensin-converting enzyme is a potent risk factor for myocardial infarction
-
Cambien F, Poirier O, Lecerf L et al. Deletion polymorphism in the gene for angiotensin-converting enzyme is a potent risk factor for myocardial infarction. Nature 1992; 359: 641-4.
-
(1992)
Nature
, vol.359
, pp. 641-644
-
-
Cambien, F.1
Poirier, O.2
Lecerf, L.3
-
11
-
-
0034606897
-
Large-scale test of hypothesised associations between the angiotensin-converting-enzyme insertion/deletion polymorphism and myocardial infarction in about 5 000 cases and 6 000 controls
-
International Studies of Infarct Survival (ISIS) Collaborators
-
Keavney B, McKenzie C, Parish S et al. Large-scale test of hypothesised associations between the angiotensin-converting-enzyme insertion/deletion polymorphism and myocardial infarction in about 5 000 cases and 6 000 controls. International Studies of Infarct Survival (ISIS) Collaborators. Lancet 2000; 355: 434-42.
-
(2000)
Lancet
, vol.355
, pp. 434-442
-
-
Keavney, B.1
McKenzie, C.2
Parish, S.3
-
13
-
-
0029738658
-
A meta-analysis of the association of the deletion allele of the angiotensin-converting enzyme gene with myocardial infarction
-
Samani NJ, Thompson JR, O'Toole L, Channer K, Woods KL. A meta-analysis of the association of the deletion allele of the angiotensin-converting enzyme gene with myocardial infarction. Circulation 1996; 94: 708-12.
-
(1996)
Circulation
, vol.94
, pp. 708-712
-
-
Samani, N.J.1
Thompson, J.R.2
O'Toole, L.3
Channer, K.4
Woods, K.L.5
-
14
-
-
0025165779
-
An insertion/deletion polymorphism in the angiotensin I-converting enzyme gene accounting for half the variance of serum enzyme levels
-
Rigat B, Hubert C, Alhenc-Gelas F, Cambien F, Corvol P, Soubrier F. An insertion/deletion polymorphism in the angiotensin I-converting enzyme gene accounting for half the variance of serum enzyme levels. J Clin Invest 1990; 86: 1343-6.
-
(1990)
J Clin Invest
, vol.86
, pp. 1343-1346
-
-
Rigat, B.1
Hubert, C.2
Alhenc-Gelas, F.3
Cambien, F.4
Corvol, P.5
Soubrier, F.6
-
15
-
-
0035572898
-
ACE genotype and endothelium-dependent vasodilation of conduit arteries and forearm microcirculation in humans
-
Arcaro G, Solini A, Monauni T et al. ACE genotype and endothelium-dependent vasodilation of conduit arteries and forearm microcirculation in humans. Arterioscler Thromb Vasc Biol 2001; 21: 1313-9.
-
(2001)
Arterioscler Thromb Vasc Biol
, vol.21
, pp. 1313-1319
-
-
Arcaro, G.1
Solini, A.2
Monauni, T.3
-
16
-
-
0032896872
-
DD angiotensin-converting enzyme gene polymorphism is associated with endothelial dysfunction in normal humans
-
Butler R, Morris AD, Burchell B, Struthers AD. DD angiotensin-converting enzyme gene polymorphism is associated with endothelial dysfunction in normal humans. Hypertension 1999; 33: 1164-8.
-
(1999)
Hypertension
, vol.33
, pp. 1164-1168
-
-
Butler, R.1
Morris, A.D.2
Burchell, B.3
Struthers, A.D.4
-
17
-
-
0031948810
-
Angiotensin-converting enzyme gene polymorphism is associated with endothelium-dependent vasodilation in never treated hypertensive patients
-
Perticone F, Ceravolo R, Maio R et al. Angiotensin-converting enzyme gene polymorphism is associated with endothelium-dependent vasodilation in never treated hypertensive patients. Hypertension 1998; 31: 900-5.
-
(1998)
Hypertension
, vol.31
, pp. 900-905
-
-
Perticone, F.1
Ceravolo, R.2
Maio, R.3
-
18
-
-
0030013235
-
The deletion polymorphism of the angiotensin-converting enzyme gene is related to phenotypic differences in human arteries
-
Buikema H, Pinto YM, Rooks G, Grandjean JG, Schunkert H, van Gilst WH. The deletion polymorphism of the angiotensin-converting enzyme gene is related to phenotypic differences in human arteries. Eur Heart J 1996; 17: 787-94.
-
(1996)
Eur Heart J
, vol.17
, pp. 787-794
-
-
Buikema, H.1
Pinto, Y.M.2
Rooks, G.3
Grandjean, J.G.4
Schunkert, H.5
Van Gilst, W.H.6
-
19
-
-
0035090199
-
Exclusion of the ACE D/I gene polymorphism as a determinant of endothelial dysfunction
-
Rossi GP, Taddei S, Virdis A et al. Exclusion of the ACE D/I gene polymorphism as a determinant of endothelial dysfunction. Hypertension 2001; 37: 293-300.
-
(2001)
Hypertension
, vol.37
, pp. 293-300
-
-
Rossi, G.P.1
Taddei, S.2
Virdis, A.3
-
20
-
-
0028092108
-
Angiotensin-converting enzyme genotype is not associated with endothelial dysfunction in subjects without other coronary risk factors
-
Celermajer DS, Sorensen KE, Barley J, Jeffrey S, Carter N, Deanfield J. Angiotensin-converting enzyme genotype is not associated with endothelial dysfunction in subjects without other coronary risk factors. Atherosclerosis 1994; 11: 121-6.
-
(1994)
Atherosclerosis
, vol.11
, pp. 121-126
-
-
Celermajer, D.S.1
Sorensen, K.E.2
Barley, J.3
Jeffrey, S.4
Carter, N.5
Deanfield, J.6
-
21
-
-
0035148502
-
Interactive Effect of Ethnicity and ACE Insertion/Deletion Polymorphism on Vascular Reactivity
-
Gainer IV, Stein CM, Neal T, Vaughan DE, Brown NJ. Interactive Effect of Ethnicity and ACE Insertion/Deletion Polymorphism on Vascular Reactivity. Hypertension 2001; 37: 46-51.
-
(2001)
Hypertension
, vol.37
, pp. 46-51
-
-
Gainer, I.V.1
Stein, C.M.2
Neal, T.3
Vaughan, D.E.4
Brown, N.J.5
-
22
-
-
0035009361
-
Polymorphisms in the renin-angiotensin system and endothelium-dependent vasodilation in normotensive subjects
-
Kurland L, Melhus H, Sarabi M, Millgard J, Ljunghall S, Lind L. Polymorphisms in the renin-angiotensin system and endothelium-dependent vasodilation in normotensive subjects. Clin Physiol 2001; 21: 343-9.
-
(2001)
Clin Physiol
, vol.21
, pp. 343-349
-
-
Kurland, L.1
Melhus, H.2
Sarabi, M.3
Millgard, J.4
Ljunghall, S.5
Lind, L.6
-
23
-
-
0032142764
-
Endothelial cell damage and angiotensin-converting enzyme insertion/deletion genotype in elderly hypertensive patients
-
Kario K, Matsuo T, Kobayashi H et al. Endothelial cell damage and angiotensin-converting enzyme insertion/deletion genotype in elderly hypertensive patients. J Am Coll Cardiol 1998; 32: 444-50.
-
(1998)
J Am Coll Cardiol
, vol.32
, pp. 444-450
-
-
Kario, K.1
Matsuo, T.2
Kobayashi, H.3
-
24
-
-
0033766782
-
ACE/DD genotype is associated with hemostasis balance disturbances reflecting hypercoagulability and endothelial dysfunction in patients with untreated hypertension
-
Makris TK, Stavroulakis GA, Dafni UG et al. ACE/DD genotype is associated with hemostasis balance disturbances reflecting hypercoagulability and endothelial dysfunction in patients with untreated hypertension. Am Heart J 2000; 140: 760-5.
-
(2000)
Am Heart J
, vol.140
, pp. 760-765
-
-
Makris, T.K.1
Stavroulakis, G.A.2
Dafni, U.G.3
-
25
-
-
0034332689
-
The insertion/deletion polymorphism of the angiotensin-converting enzyme gene determines coronary vascular tone and nitric oxide activity
-
Prasad A, Narayanan S, Waclawiw MA, Estein N, Quyyumi AA. The insertion/deletion polymorphism of the angiotensin-converting enzyme gene determines coronary vascular tone and nitric oxide activity. J Am Coll Cardiol 2000; 36: 1579-86.
-
(2000)
J Am Coll Cardiol
, vol.36
, pp. 1579-1586
-
-
Prasad, A.1
Narayanan, S.2
Waclawiw, M.A.3
Estein, N.4
Quyyumi, A.A.5
-
26
-
-
0242668430
-
DD ACE gene polymorphism is associated with increased coronary artery endothelial dysfunction: The PREFACE trial
-
Mulder HJ, van Geel PP, Schalij MJ, van Gilst WH, Zwinderman AH, Bruschke AV. DD ACE gene polymorphism is associated with increased coronary artery endothelial dysfunction: the PREFACE trial. Heart 2003; 89: 557-8.
-
(2003)
Heart
, vol.89
, pp. 557-558
-
-
Mulder, H.J.1
Van Geel, P.P.2
Schalij, M.J.3
Van Gilst, W.H.4
Zwinderman, A.H.5
Bruschke, A.V.6
-
27
-
-
0034332774
-
Contribution of bradykinin receptor dysfunction to abnormal coronary vasomotion in humans
-
Prasad A, Husain S, Schenke W, Mincemoyer R, Epstein N, Quyyumi AA. Contribution of bradykinin receptor dysfunction to abnormal coronary vasomotion in humans. J Am Coll Cardiol 2000; 36: 1467-73.
-
(2000)
J Am Coll Cardiol
, vol.36
, pp. 1467-1473
-
-
Prasad, A.1
Husain, S.2
Schenke, W.3
Mincemoyer, R.4
Epstein, N.5
Quyyumi, A.A.6
-
28
-
-
0034604252
-
Insertion-deletion polymorphism of the ACE gene modulates reversibility of endothelial dysfunction with ACE inhibition
-
Prasad A, Narayanan S, Husain S et al. Insertion-deletion polymorphism of the ACE gene modulates reversibility of endothelial dysfunction with ACE inhibition. Circulation 2000; 102: 35-41.
-
(2000)
Circulation
, vol.102
, pp. 35-41
-
-
Prasad, A.1
Narayanan, S.2
Husain, S.3
-
29
-
-
0034097536
-
Angiotensin II type 1 receptor A1166C gene polymorphism is associated with an increased response to angiotensin II in human arteries
-
Van Gel PP, Pinto YM, Voors AA et al. Angiotensin II type 1 receptor A1166C gene polymorphism is associated with an increased response to angiotensin II in human arteries. Hypertension 2000; 35: 717-21.
-
(2000)
Hypertension
, vol.35
, pp. 717-721
-
-
Van Gel, P.P.1
Pinto, Y.M.2
Voors, A.A.3
-
30
-
-
0025277646
-
Control of coronary vascular tone by nitric oxide
-
Kelm M, Schrader J. Control of coronary vascular tone by nitric oxide. Circ Res 1990; 66: 1561-75.
-
(1990)
Circ Res
, vol.66
, pp. 1561-1575
-
-
Kelm, M.1
Schrader, J.2
-
31
-
-
0033535841
-
T-786→C mutation in the 5′-flanking region of the endothelial nitric oxide synthase gene is associated with coronary spasm
-
Nakayama M, Yasue H, Yoshimura M et al. T-786→C mutation in the 5′-flanking region of the endothelial nitric oxide synthase gene is associated with coronary spasm. Circulation 1999; 99: 2864-70.
-
(1999)
Circulation
, vol.99
, pp. 2864-2870
-
-
Nakayama, M.1
Yasue, H.2
Yoshimura, M.3
-
32
-
-
0036020984
-
Effects of endothelial nitric oxide synthase gene polymorphisms on platelet function, nitric oxide release, and interactions with estradiol
-
Tanus-Santos JE, Desai M, Deak LR et al. Effects of endothelial nitric oxide synthase gene polymorphisms on platelet function, nitric oxide release, and interactions with estradiol. Pharmacogenetics 2002; 12: 407-13.
-
(2002)
Pharmacogenetics
, vol.12
, pp. 407-413
-
-
Tanus-Santos, J.E.1
Desai, M.2
Deak, L.R.3
-
33
-
-
0037372779
-
Endothelial nitric oxide synthase gene polymorphisms and risk of coronary artery disease
-
Colombo MG, Paradossi U, Andreassi MG et al. Endothelial nitric oxide synthase gene polymorphisms and risk of coronary artery disease. Clin Chem 2003; 49: 389-95.
-
(2003)
Clin Chem
, vol.49
, pp. 389-395
-
-
Colombo, M.G.1
Paradossi, U.2
Andreassi, M.G.3
-
34
-
-
0036900387
-
The endothelial nitric oxide synthase (Glu298Asp and -786T>C) gene polymorphisms are associated with coronary in-stent restenosis
-
Gomma AH, Elrayess MA, Knight CJ, Hawe E, Fox KM, Humphries SE. The endothelial nitric oxide synthase (Glu298Asp and -786T>C) gene polymorphisms are associated with coronary in-stent restenosis. Eur Heart J 2002; 23: 1955-62.
-
(2002)
Eur Heart J
, vol.23
, pp. 1955-1962
-
-
Gomma, A.H.1
Elrayess, M.A.2
Knight, C.J.3
Hawe, E.4
Fox, K.M.5
Humphries, S.E.6
-
35
-
-
0034652823
-
A T-786→C mutation in the 5′-flanking region of the endothelial nitric oxide synthase gene and coronary arterial vasomotility
-
Yoshimura M, Nakayama M, Shimasaki Y et al. A T-786→C mutation in the 5′-flanking region of the endothelial nitric oxide synthase gene and coronary arterial vasomotility. Am J Cardiol 2000; 85: 710-4.
-
(2000)
Am J Cardiol
, vol.85
, pp. 710-714
-
-
Yoshimura, M.1
Nakayama, M.2
Shimasaki, Y.3
-
36
-
-
0037454141
-
The T-786C and Glu298Asp polymorphisms of the endothelial nitric oxide gene affect the forearm blood flow responses of Caucasian hypertensive patients
-
Rossi GP, Taddei S, Virdis A et al. The T-786C and Glu298Asp polymorphisms of the endothelial nitric oxide gene affect the forearm blood flow responses of Caucasian hypertensive patients. J Am Coll Cardiol 2003; 41: 938-45.
-
(2003)
J Am Coll Cardiol
, vol.41
, pp. 938-945
-
-
Rossi, G.P.1
Taddei, S.2
Virdis, A.3
-
37
-
-
1842573032
-
Relevance of the T-786C and Glu298Asp variants in the endothelial nitric oxide synthase gene for cholinergic and adrenergic coronary vasomotor responses in man
-
Abstract
-
CK Naber, O Oldenburg, U Frey et al. Relevance of the T-786C and Glu298Asp variants in the endothelial nitric oxide synthase gene for cholinergic and adrenergic coronary vasomotor responses in man. Circulation 2003; 106 (Suppl): 1042 (Abstract).
-
(2003)
Circulation
, vol.106
, Issue.SUPPL.
, pp. 1042
-
-
Naber, C.K.1
Oldenburg, O.2
Frey, U.3
-
38
-
-
0035854834
-
Acidic hydrolysis as a mechanism for the cleavage of the Glu(298)→Asp variant of human endothelial nitric-oxide synthase
-
Fairchild TA, Fulton D, Fontana JT, Gratton JP, McCabe TJ, Sessa WC. Acidic hydrolysis as a mechanism for the cleavage of the Glu(298)→Asp variant of human endothelial nitric-oxide synthase. J Biol Chem 2001; 276: 26674-9.
-
(2001)
J Biol Chem
, vol.276
, pp. 26674-26679
-
-
Fairchild, T.A.1
Fulton, D.2
Fontana, J.T.3
Gratton, J.P.4
McCabe, T.J.5
Sessa, W.C.6
-
39
-
-
0034646248
-
Intracellular processing of endothelial nitric oxide synthase isoforms associated with differences in severity of cardiopulmonary at diseases: Cleavage of proteins with aspartate vs. glutamate at position 298
-
Tesauro M, Thampson WC, Rogliani P, Qi L, Chaudhary PP, Moss J. Intracellular processing of endothelial nitric oxide synthase isoforms associated with differences in severity of cardiopulmonary at diseases: cleavage of proteins with aspartate vs. glutamate at position 298. Proc Natl Acad Sci U S A 2000; 97: 2832-5.
-
(2000)
Proc Natl Acad Sci U S A
, vol.97
, pp. 2832-2835
-
-
Tesauro, M.1
Thampson, W.C.2
Rogliani, P.3
Qi, L.4
Chaudhary, P.P.5
Moss, J.6
-
40
-
-
0345192295
-
A common variant of the endothelial nitric oxide synthase (Glu298→Asp) is a major risk factor for coronary artery disease in the UK
-
Hingorani AD, Liang CF, Fatibene J et al. A common variant of the endothelial nitric oxide synthase (Glu298→Asp) is a major risk factor for coronary artery disease in the UK. Circulation 1999; 100: 1515-20.
-
(1999)
Circulation
, vol.100
, pp. 1515-1520
-
-
Hingorani, A.D.1
Liang, C.F.2
Fatibene, J.3
-
41
-
-
0031902667
-
Endothelial nitric oxide synthase gene is positively associated with essential hypertension
-
Miyamoto Y, Saito Y, Kajiyama N et al. Endothelial nitric oxide synthase gene is positively associated with essential hypertension. Hypertension 1998; 32: 3-8.
-
(1998)
Hypertension
, vol.32
, pp. 3-8
-
-
Miyamoto, Y.1
Saito, Y.2
Kajiyama, N.3
-
42
-
-
0031748762
-
Association of the missense Glu298Asp variant of the endothelial nitric oxide synthase gene with myocardial infarction
-
Shimasaki Y, Yasue H, Yoshimura M et al. Association of the missense Glu298Asp variant of the endothelial nitric oxide synthase gene with myocardial infarction. J Am Coll Cardiol 1998; 31: 1506-10.
-
(1998)
J Am Coll Cardiol
, vol.31
, pp. 1506-1510
-
-
Shimasaki, Y.1
Yasue, H.2
Yoshimura, M.3
-
43
-
-
0035205189
-
eNOS 894T allele and coronary blood flow at rest and during adenosine-induced hyperemia
-
Naber CK, Baumgart D, Altmann C, Siffert W, Erbel R, Heusch G. eNOS 894T allele and coronary blood flow at rest and during adenosine-induced hyperemia. Am J Physiol Heart Circ Physiol 2001; 281: H1908-H1912.
-
(2001)
Am J Physiol Heart Circ Physiol
, vol.281
-
-
Naber, C.K.1
Baumgart, D.2
Altmann, C.3
Siffert, W.4
Erbel, R.5
Heusch, G.6
-
44
-
-
0035932773
-
Relationship between the G894T polymorphism (Glu298Asp variant) in endothelial nitric oxide synthase and nitric oxide-mediated endothelial function in human atherosclerosis
-
Guzik TJ, Black E, West NE et al. Relationship between the G894T polymorphism (Glu298Asp variant) in endothelial nitric oxide synthase and nitric oxide-mediated endothelial function in human atherosclerosis. Am J Med Genet 2001; 100: 130-7.
-
(2001)
Am J Med Genet
, vol.100
, pp. 130-137
-
-
Guzik, T.J.1
Black, E.2
West, N.E.3
-
45
-
-
0037076794
-
Glu298Asp endothelial nitric oxide synthase gene polymorphism interacts with environmental and dietary factors to influence endothelial function
-
Leeson CP, Hingorani AD, Mullen MJ et al. Glu298Asp endothelial nitric oxide synthase gene polymorphism interacts with environmental and dietary factors to influence endothelial function. Circ Res 2002; 90: 1153-8.
-
(2002)
Circ Res
, vol.90
, pp. 1153-1158
-
-
Leeson, C.P.1
Hingorani, A.D.2
Mullen, M.J.3
-
46
-
-
0033669591
-
Functional gene testing of the Glu298Asp polymorphism of the endothelial NO synthase
-
Schneider MP, Erdmann J, Delles C, Fleck E, Regitz-Zagrosek V, Schmieder RE. Functional gene testing of the Glu298Asp polymorphism of the endothelial NO synthase. J Hypertens 2000; 18: 1767-73.
-
(2000)
J Hypertens
, vol.18
, pp. 1767-1773
-
-
Schneider, M.P.1
Erdmann, J.2
Delles, C.3
Fleck, E.4
Regitz-Zagrosek, V.5
Schmieder, R.E.6
-
47
-
-
0037454272
-
The T-786C endothelial nitric oxide synthase genotype is a novel risk factor for coronary artery disease in Caucasian patients of the GENICA study
-
Rossi GP, Cesari M, Zanchetta M et al. The T-786C endothelial nitric oxide synthase genotype is a novel risk factor for coronary artery disease in Caucasian patients of the GENICA study. J Am Coll Cardiol 2003; 41: 930-7.
-
(2003)
J Am Coll Cardiol
, vol.41
, pp. 930-937
-
-
Rossi, G.P.1
Cesari, M.2
Zanchetta, M.3
-
48
-
-
0035174449
-
Effects of ethnicity on the distribution of clinically relevant endothelial nitric oxide variants
-
Tanus-Santos JE, Desai M, Flockhart DA. Effects of ethnicity on the distribution of clinically relevant endothelial nitric oxide variants. Pharmacogenetics 2001; 11: 719-25.
-
(2001)
Pharmacogenetics
, vol.11
, pp. 719-725
-
-
Tanus-Santos, J.E.1
Desai, M.2
Flockhart, D.A.3
-
49
-
-
0034768750
-
Association analyses between genetic polymorphisms of endothelial nitric oxide synthase gene and hypertension in Japanese: The Suita Study
-
Tsujita Y, Baba S, Yamauchi R et al. Association analyses between genetic polymorphisms of endothelial nitric oxide synthase gene and hypertension in Japanese: The Suita Study. J Hypertens 2001; 19: 1941-8.
-
(2001)
J Hypertens
, vol.19
, pp. 1941-1948
-
-
Tsujita, Y.1
Baba, S.2
Yamauchi, R.3
-
50
-
-
0033608950
-
Augmented alpha-adrenergic constriction of atherosclerotic human coronary arteries
-
Baumgart D, Haude M, Gorge G et al. Augmented alpha-adrenergic constriction of atherosclerotic human coronary arteries. Circulation 1999; 99: 2090-7.
-
(1999)
Circulation
, vol.99
, pp. 2090-2097
-
-
Baumgart, D.1
Haude, M.2
Gorge, G.3
-
51
-
-
0037440041
-
Variation in the alpha2B-adrenoceptor gene as a risk factor for prehospital fatal myocardial infarction and sudden cardiac death
-
Snapir A, Mikkelsson J, Perola M, Penttila A, Scheinin M, Karhunen PJ. Variation in the alpha2B-adrenoceptor gene as a risk factor for prehospital fatal myocardial infarction and sudden cardiac death. J Am Coll Cardiol 2003; 41: 190-4.
-
(2003)
J Am Coll Cardiol
, vol.41
, pp. 190-194
-
-
Snapir, A.1
Mikkelsson, J.2
Perola, M.3
Penttila, A.4
Scheinin, M.5
Karhunen, P.J.6
-
52
-
-
0035028708
-
An insertion/deletion polymorphism in the alpha2B-adrenergic receptor gene is a novel genetic risk factor for acute coronary events
-
Snapir A, Heinonen P, Tuomainen TP et al. An insertion/deletion polymorphism in the alpha2B-adrenergic receptor gene is a novel genetic risk factor for acute coronary events. J Am Coll Cardiol 2001; 37: 1516-22.
-
(2001)
J Am Coll Cardiol
, vol.37
, pp. 1516-1522
-
-
Snapir, A.1
Heinonen, P.2
Tuomainen, T.P.3
-
53
-
-
0038017249
-
Effects of common polymorphisms in the alpha 1A-, alpha2B-, beta1- and beta2-adrenoreceptors on haemodynamic responses to adrenaline
-
Snapir A, Koskenvuo J, Taikka J et al. Effects of common polymorphisms in the alpha 1A-, alpha2B-, beta1- and beta2-adrenoreceptors on haemodynamic responses to adrenaline. Clin Sci (Lond) 2003; 104: 509-20.
-
(2003)
Clin Sci (Lond)
, vol.104
, pp. 509-520
-
-
Snapir, A.1
Koskenvuo, J.2
Taikka, J.3
-
54
-
-
0036844855
-
Deletion polymorphism in the alpha2B-adrenergic receptor gene is associated with flow-mediated dilatation of the brachial artery
-
Heinonen P, Jartti L, Jarvisalo MJ et al. Deletion polymorphism in the alpha2B-adrenergic receptor gene is associated with flow-mediated dilatation of the brachial artery. Clin Sci (Lond) 2002; 103: 517-24.
-
(2002)
Clin Sci (Lond)
, vol.103
, pp. 517-524
-
-
Heinonen, P.1
Jartti, L.2
Jarvisalo, M.J.3
-
55
-
-
0032589708
-
G protein beta3 subunit 825T allele and enhanced coronary vasoconstriction on algha(2)-adrenoceptor activation
-
Baumgart D, Naber C, Haude M et al. G protein beta3 subunit 825T allele and enhanced coronary vasoconstriction on algha(2)-adrenoceptor activation. Circ Res 1997; 85: 965-9.
-
(1997)
Circ Res
, vol.85
, pp. 965-969
-
-
Baumgart, D.1
Naber, C.2
Haude, M.3
-
56
-
-
17344366286
-
Association of a human G-protein beta3 subunit variant with hypertension
-
Siffert W, Rosskopf D, Siffert G et al. Association of a human G-protein beta3 subunit variant with hypertension. Nat Genet 1998; 18: 45-8.
-
(1998)
Nat Genet
, vol.18
, pp. 45-48
-
-
Siffert, W.1
Rosskopf, D.2
Siffert, G.3
-
57
-
-
0034634513
-
Enhanced epinephrine-induced platelet aggregation in individuals carrying the G protein beta3 subunit 825T allele
-
Naber C, Hermann BL, Vietzke D et al. Enhanced epinephrine-induced platelet aggregation in individuals carrying the G protein beta3 subunit 825T allele. FEBS Lett 2000; 484: 199-201.
-
(2000)
FEBS Lett
, vol.484
, pp. 199-201
-
-
Naber, C.1
Hermann, B.L.2
Vietzke, D.3
-
58
-
-
0027214241
-
The release of endothelium-derived relaxing factor via alpha 2-adrenergic-receptor activation is specifically mediated by Gi alpha 2
-
Liao X, Homey O. The release of endothelium-derived relaxing factor via alpha 2-adrenergic-receptor activation is specifically mediated by Gi alpha 2. J Biol Chem 1993; 268: 19528-33.
-
(1993)
J Biol Chem
, vol.268
, pp. 19528-19533
-
-
Liao, X.1
Homey, O.2
-
59
-
-
0037565530
-
Role of the eNOS Glu298Asp variant an the GNB3825T allele dependent determination of alpha-adrenergic caronary constriction
-
Naber CK, Baumgart D, Heusch G et al. Role of the eNOS Glu298Asp variant an the GNB3825T allele dependent determination of alpha-adrenergic caronary constriction. Pharmacogenetics 2003; 13: 279-84.
-
(2003)
Pharmacogenetics
, vol.13
, pp. 279-284
-
-
Naber, C.K.1
Baumgart, D.2
Heusch, G.3
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