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Volumn 112, Issue 1-4, 2002, Pages 39-49

Aberrant splicing of FHIT transcripts in human gastric cancer cell lines

Author keywords

Aberrant FHIT transcripts; Downstream splice processing; Exonic deletions; FHIT gene; Gastric cancer; Gene alterations; Human cancers; Initiation codon; Intron 5 sequences; Multi step splice patterns; Reverse transcription polymerase chain reaction

Indexed keywords

FRAGILE HISTIDINE TRIAD PROTEIN;

EID: 1542636403     PISSN: 10780297     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article
Times cited : (7)

References (12)
  • 4
    • 0037096934 scopus 로고    scopus 로고
    • Identification of unstable sequence within the common fragile site at 3p14.2: Implications for the mechanism of deletions within fragile histidine triad gene/common fragile site at 3p14.2 in tumors
    • Corbin, S., Neilly, M.E., Espinosa, R. 3rd., Davis, E. M., McKeithan, T. W. and Le Beau, M. M. (2002). Identification of unstable sequence within the common fragile site at 3p14.2: implications for the mechanism of deletions within fragile histidine triad gene/common fragile site at 3p14.2 in tumors. Cancer Res. 62: 3477-3484.
    • (2002) Cancer Res. , vol.62 , pp. 3477-3484
    • Corbin, S.1    Neilly, M.E.2    Espinosa III, R.3    Davis, E.M.4    McKeithan, T.W.5    Le Beau, M.M.6
  • 6
    • 0030593719 scopus 로고    scopus 로고
    • Analysis of possible WT1 RNA processing in primary Wilms tumors
    • Gunning, K. B., Cohn, S. L., Tomlinson, G. E., Strong, L. C. and Huff, V. (1996). Analysis of possible WT1 RNA processing in primary Wilms tumors. Oncogene 13:1179-1185.
    • (1996) Oncogene , vol.13 , pp. 1179-1185
    • Gunning, K.B.1    Cohn, S.L.2    Tomlinson, G.E.3    Strong, L.C.4    Huff, V.5
  • 7
    • 0033565920 scopus 로고    scopus 로고
    • Induction of apoptosis and inhibition of tumorigenicity and tumor growth by adenovirus vector-mediated fragile histidine triad (FHIT) gene overexpression
    • Ji, L., Fang, B., Yen, N., Fong, K., Minna, J. D. and Roth, J. A. (1999). Induction of apoptosis and inhibition of tumorigenicity and tumor growth by adenovirus vector-mediated fragile histidine triad (FHIT) gene overexpression. Cancer Res. 59: 3333-3339.
    • (1999) Cancer Res. , vol.59 , pp. 3333-3339
    • Ji, L.1    Fang, B.2    Yen, N.3    Fong, K.4    Minna, J.D.5    Roth, J.A.6
  • 9
    • 13344279424 scopus 로고    scopus 로고
    • The FHIT gene, spanning the chromosome 3p14.2 fragile site and renal carcinoma-associated t(3;8) breakpoint, is abnormal in digestive tract cancers
    • Ohta, M., Inoue, H., Cotticelli, M. G., Kastury, K., Baffa, R., Palazzo, J., Siprashvili, Z., Mori, M., McCue, P., Druck, T., Croce, C. M. and Huebner, K. (1996). The FHIT gene, spanning the chromosome 3p14.2 fragile site and renal carcinoma-associated t(3;8) breakpoint, is abnormal in digestive tract cancers. Cell 84: 587-597.
    • (1996) Cell , vol.84 , pp. 587-597
    • Ohta, M.1    Inoue, H.2    Cotticelli, M.G.3    Kastury, K.4    Baffa, R.5    Palazzo, J.6    Siprashvili, Z.7    Mori, M.8    McCue, P.9    Druck, T.10    Croce, C.M.11    Huebner, K.12


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.