Neurological Disorder after Borna Disease Virus Infection in the Absence of Either Interferon-γ, Fas, Inducible NO Synthase, or Chemokine Receptor CXCR3

Viral Immunology

Volumn 17, Issue 1, 2004, Pages 79-85

  Hausmann, Jürgen ^a   Sauder, Christian ^a   Wasmer, Mathias ^a   Lu, Bao ^b   Staeheli, Peter ^a  

^a UNIVERSITY OF FREIBURG   (Germany)

^b BOSTON CHILDREN S HOSPITAL   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CHEMOKINE RECEPTOR CCR10; CHEMOKINE RECEPTOR CXCR3; FAS ANTIGEN; GAMMA INTERFERON; N6 (1 IMINOETHYL)LYSINE; NITRIC OXIDE SYNTHASE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; ASTROCYTE; BORNA DISEASE; BORNA DISEASE VIRUS; BRAIN INFECTION; CELL FUNCTION; CONTROLLED STUDY; DRUG MECHANISM; ENZYME ACTIVITY; IMMUNE RESPONSE; IMMUNOPATHOLOGY; KINETICS; MENINGOENCEPHALITIS; MOUSE; NEUROLOGIC DISEASE; NONHUMAN; RAT STRAIN; T LYMPHOCYTE; TRANSGENIC MOUSE; VIRUS PATHOGENESIS; VIRUS VIRULENCE;

ANIMALS; ANTIGENS, CD95; BORNA DISEASE; BORNA DISEASE VIRUS; CD8-POSITIVE T-LYMPHOCYTES; ENZYME INDUCTION; INTERFERON TYPE II; MICE; MICE, KNOCKOUT; NERVOUS SYSTEM DISEASES; NITRIC OXIDE SYNTHASE; RATS; RATS, INBRED LEW; RECEPTORS, CHEMOKINE;

ANIMALIA; BORNA DISEASE VIRUS; MUS MUSCULUS;

EID: 1542513305     PISSN: 08828245     EISSN: None     Source Type: Journal    
DOI: 10.1089/088282404322875476     Document Type: Article

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