Induction of serine racemase by inflammatory stimuli is dependent on AP-1

Annals of the New York Academy of Sciences

Volumn 1035, Issue , 2004, Pages 133-146

  Wu, Shengzhou ^a   Barger, Steven W ^a,b,c  

^a UNIVERSITY OF ARKANSAS FOR MEDICAL SCIENCES   (United States)

^b UNIVERSITY OF ARKANSAS FOR MEDICAL SCIENCES   (United States)

^c GERIATRIC RESEARCH EDUCATION AND CLINICAL CENTER   (United States)

Author keywords

D serine; Dexamethasone; Gene regulation; JNK; Lipopolysaccharide; Transcription

Indexed keywords

AMYLOID BETA PROTEIN; DEXAMETHASONE; GENOMIC DNA; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; LIPOPOLYSACCHARIDE; LUCIFERASE; MESSENGER RNA; N METHYL DEXTRO ASPARTIC ACID RECEPTOR STIMULATING AGENT; PYRUVIC ACID; RACEMASE; SERINE; SERINE RACEMASE; STRESS ACTIVATED PROTEIN KINASE; TRANSCRIPTION FACTOR AP 1; UNCLASSIFIED DRUG;

ANIMAL CELL; ASTROCYTOMA CELL; BINDING SITE; CONFERENCE PAPER; CONTROLLED STUDY; DEGENERATIVE DISEASE; ENZYME ACTIVITY; ENZYME INDUCTION; GEL MOBILITY SHIFT ASSAY; HUMAN; HUMAN CELL; MICROGLIA; MOUSE; NERVOUS SYSTEM INFLAMMATION; NONHUMAN; ONCOGENE C FOS; PROMOTER REGION; REAL TIME POLYMERASE CHAIN REACTION; SIGNAL TRANSDUCTION; SITE DIRECTED MUTAGENESIS; TRANSCRIPTION REGULATION; WESTERN BLOTTING;

EID: 14944354601     PISSN: 00778923     EISSN: None     Source Type: Book Series    
DOI: 10.1196/annals.1332.009     Document Type: Conference Paper

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