Rebuttal to Bredesen: 'The non-existent aging program: How does it work?'

Aging Cell

Volumn 3, Issue 5, 2004, Pages 253-254

  Austad, Steven N ^a  

^a UNIVERSITY OF TEXAS HEALTH SCIENCE CENTER AT SAN ANTONIO   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

TESTOSTERONE;

AGING; APOPTOSIS; ARTICLE; BIOCHEMISTRY; CAENORHABDITIS ELEGANS; CASTRATION; CAT; CELL ACTIVITY; CLINICAL RESEARCH; DEATH; DIETARY INTAKE; DOG; ENVIRONMENTAL FACTOR; EVOLUTION; GENE EXPRESSION REGULATION; GENE FUNCTION; GENE INACTIVATION; GENETIC ANALYSIS; GERONTOLOGY; HUMAN; LONGEVITY; MOLECULAR BIOLOGY; MOUSE; NATURAL SELECTION; NONHUMAN; OBSERVATION; PHENOTYPE; PHENOTYPIC VARIATION; PHYSICAL ACTIVITY; PRIORITY JOURNAL; SALMON; SENESCENCE; SIGNAL TRANSDUCTION; SPECIES DIFFERENCE; STEREOTYPY; TESTOSTERONE SYNTHESIS; THEORY;

AGE FACTORS; AGING; ANIMALS; HUMANS; MUTATION;

EID: 13844300060     PISSN: 14749718     EISSN: None     Source Type: Journal    
DOI: 10.1111/j.1474-9728.2004.00119.x     Document Type: Article

References (4)

1. Finch CE, Kirkwood TRL (2000) Chance, Development and Aging. New York: Oxford University Press.
2. Herndon LA, Schmeissner PJ, Dudaronek JM, Brown PA, Listner KM, Sakana Y, Paupard MC, Hall DH, Driscoll M (2002) Stochastic and genetic factors influence tissue-specific decline in ageing C. elegans. Nature 419, 808-814.
3. Promislow DEL (2004) Protein networks, pleitropy and the evolution of senescence. Proc. R. Soc. Lond. B Bio. Sci. 271, 1225-1234.
4. Walker DW, McColl G, Jenkins NL, Harris H, Lithgow GJ (2000) Natural selection: evolution of lifespan in C. elegans. Nature 405, 296-297.