Na/Ca Exchanger Overexpression Induces Endoplasmic Reticulum Stress, Caspase-12 Release, and Apoptosis

Annals of the New York Academy of Sciences

Volumn 1010, Issue , 2003, Pages 430-432

  Díaz Horta, Oscar ^a,b   Van Eylen, Françoise ^a   Herchuelz, André ^a,c  

^a FREE UNIVERSITY OF BRUSSELS   (Belgium)

^b INSTITUTO NACIONAL DE ENDOCRINOLOGÍA   (Cuba)

^c UNIVERSITÉ LIBRE DE BRUXELLES   (Belgium)

Author keywords

Apoptosis; Caspase 12; Endoplasmic reticulum; Na Ca exchanger overexpression

Indexed keywords

CALCIUM ION; CASPASE 12; SODIUM CALCIUM EXCHANGE PROTEIN;

APOPTOSIS; CALCIUM HOMEOSTASIS; CONFERENCE PAPER; ENDOPLASMIC RETICULUM; ENZYME RELEASE; PANCREAS ISLET BETA CELL; PROTEIN EXPRESSION; SODIUM CALCIUM EXCHANGE; STRESS;

EID: 1342284269     PISSN: 00778923     EISSN: None     Source Type: Book Series    
DOI: 10.1196/annals.1299.077     Document Type: Conference Paper

References (2)

1. NICOTERA, P. & S. ORRENIUS. 1998. The role of calcium in apoptosis. Cell Calcium 213: 173-180.
2. NAKAGAWA, T., H. ZHU, N. MORISHIMA, et al. 2000. Caspase-12 mediates endoplasmic-reticulum-specific apoptosis and cytotoxicity by amyloid-β. Nature 403: 98-103.