Sudden cardiac death: A matter of faulty ion channels?

Current Biology

Volumn 8, Issue 16, 1998, Pages

  Jongsma, H J ^a  

^a UTRECHT UNIVERSITY   (Netherlands)

Author keywords

[No Author keywords available]

Indexed keywords

EID: 13144282671     PISSN: 09609822     EISSN: None     Source Type: Journal    
DOI: 10.1016/s0960-9822(07)00363-6     Document Type: Article

References (10)

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4. Wang Q, Curran ME, Splawski I, Burn TC, Milholland JM, VanRaay TJ, Shen J, Timothy KW, Vincent GM, de Jager T, et al.: Positional cloning of a novel potassium channel gene: KVLQT1 mutations cause cardiac arrhythmias. Nat Genet 1996, 12:17-23.
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6. + channels expressed in mammalian cells. Am J Physiol 1998, 274:H1643-H1654.
7. + channel dysfunction in an inherited cardiac arrhythmia. Proc Natl Acad Sci USA 1996, 93:2208-2212.
8. Shalaby FY, Levesque PC, Yang WP, Little WA, Conder ML, Jenkins-West T, Blanar MA: Dominant-negative KvLQT1 mutations underlie the LQT1 form of long QT syndrome. Circulation 1997, 96:1733-1736.
9. London B, Jeron A, Zhou J, Buckett P, Han X, Mitchell GF, Koren G: Long QT and ventricular arrhythmias in transgenic mice expressing the N terminus and first transmembrane segment of a voltage-gated potassium channel. Proc Natl Acad Sci USA 1998, 95:2926-2931.
10. Chen Q, Kirsch GE, Zhang D, Brugada R, Brugada P, Brugada J, Potenza D, Moya A, Borggrefe M, Breithardt G, et al.: Genetic basis and molecular mechanism for idiopathic ventricular fibrillation. Nature 1998, 392:293-296.