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Volumn 96, Issue 3, 2004, Pages 276-279

Molecular mechanisms and drug development in aquaporin water channel diseases: Aquaporin superfamily (superaquaporins): Expansion of aquaporins restricted to multicellular organisms

Author keywords

Aquaporin; Asparagine proline alanine (NPA) box; Knockout mice; Polycystic kidney

Indexed keywords

ALANINE; AQUAPORIN; ASPARAGINE; PROLINE;

EID: 10944255127     PISSN: 13478613     EISSN: None     Source Type: Journal    
DOI: 10.1254/jphs.FMJ04004X7     Document Type: Short Survey
Times cited : (114)

References (10)
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  • 2
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  • 4
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    • Homology modeling of representative subfamilies of Arabidopsis major intrinsic proteins. Classification based on the aromatic/arginine selectivity filter
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    • Wallace, I.S.1    Roberts, D.M.2
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  • 8
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    • Aquaporin-0 membrane junctions reveal the structure of a closed water pore
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    • Gonen, T.1    Sliz, P.2    Kistler, J.3    Cheng, Y.4    Walz, T.5
  • 9
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    • Neonatal mortality of aquaporin 11 knockout mice: A novel model for autosomal recessive polycystic kidney disease
    • San Diego
    • Morishita Y, Ishibashi K, Ando Y, Muto S, Sasaki S, Kusano E. Neonatal mortality of aquaporin 11 knockout mice: a novel model for autosomal recessive polycystic kidney disease. ASN Annual Meeting, San Diego, 2003.
    • (2003) ASN Annual Meeting
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    • Mutations in a novel gene, NPHP3, cause adolescent nephronophthisis, tapeto-retinal degeneration and hepatic fibrosis
    • Olbrich H, Fliegauf M, Hoefele J, Kispert A, Otto E, Volz A, et al. Mutations in a novel gene, NPHP3, cause adolescent nephronophthisis, tapeto-retinal degeneration and hepatic fibrosis. Nat Genet. 2003;34:455-459.
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.