Normal Cells Control the Growth of Neighboring Transformed Cells Independent of Gap Junctional Communication and Src Activity

Cancer Research

Volumn 64, Issue 4, 2004, Pages 1347-1358

  Alexander, David B ^a   Ichikawa, Hitoshi ^b   Bechberger, John F ^c   Valiunas, Virginijus ^a   Ohki, Misao ^b   Naus, Christian C G ^c   Kunimoto, Takehiko ^b   Tsuda, Hiroyuki ^b   Miller, W Todd ^a   Goldberg, Gary S ^a  

^a STONY BROOK UNIVERSITY   (United States)

^b NATIONAL CANCER CENTER RESEARCH INSTITUTE   (Japan)

^c UNIVERSITY OF BRITISH COLUMBIA   (Canada)

Author keywords

[No Author keywords available]

Indexed keywords

LIM PROTEIN; MITOGEN ACTIVATED PROTEIN KINASE INHIBITOR; PROTEIN; PROTEIN SERINE KINASE; SERUM DEPRIVATION RESPONSE PROTEIN; SOMATOMEDIN C; TRANSCRIPTION FACTOR; UNCLASSIFIED DRUG; WNT PROTEIN;

ANIMAL CELL; ARTICLE; CANCER CELL; CANCER CELL CULTURE; CELL COMMUNICATION; CELL CONTACT; CELL GROWTH; CELL JUNCTION; CELL TRANSFORMATION; CONTROLLED STUDY; ENZYME ACTIVITY; GAP JUNCTION; GROWTH INHIBITION; GROWTH REGULATION; MOUSE; NONHUMAN; NUCLEOTIDE SEQUENCE; PRIORITY JOURNAL; PROTEIN EXPRESSION; REGULATORY MECHANISM;

ANIMALS; CELL COMMUNICATION; CELL DIVISION; CELL TRANSFORMATION, NEOPLASTIC; CONNEXIN 43; GAP JUNCTIONS; GENE EXPRESSION REGULATION; MICE; MICE, KNOCKOUT; SRC-FAMILY KINASES; TRANSCRIPTION, GENETIC;

EID: 10744224741     PISSN: 00085472     EISSN: None     Source Type: Journal    
DOI: 10.1158/0008-5472.CAN-03-2558     Document Type: Article

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