Severe Parkinson's disease in rheumatoid arthritis patient treated with infliximab [8]

Rheumatology

Volumn 42, Issue 5, 2003, Pages 702-703

  Hrycaj, P ^a   Korczowska, I ^a   Lacki J K ^a  

^a POZNAN UNIVERSITY OF MEDICAL SCIENCES   (Poland)

Author keywords

[No Author keywords available]

Indexed keywords

BENSERAZIDE; INFLIXIMAB; LEVODOPA; METHOTREXATE; NONSTEROID ANTIINFLAMMATORY AGENT; PREDNISONE; RHEUMATOID FACTOR; TUMOR NECROSIS FACTOR ALPHA; TUMOR NECROSIS FACTOR ALPHA CONVERTING ENZYME INHIBITOR; TUMOR NECROSIS FACTOR ALPHA RECEPTOR; ANTIRHEUMATIC AGENT; MONOCLONAL ANTIBODY;

AGED; BREATHING DISORDER; CASE REPORT; CLINICAL FEATURE; DIAGNOSTIC TEST; DISEASE SEVERITY; DOPAMINERGIC NERVE CELL; DOSE RESPONSE; DYSPHAGIA; FEMALE; HAND RADIOGRAPHY; HUMAN; LETTER; LOW DRUG DOSE; PARKINSON DISEASE; POLYARTHRITIS; PRIORITY JOURNAL; REMISSION; RHEUMATOID ARTHRITIS; RIGIDITY; SPEECH DISORDER; TREMOR; CHEMICALLY INDUCED DISORDER; DRUG ANTAGONISM;

AGED; ANTIBODIES, MONOCLONAL; ANTIRHEUMATIC AGENTS; ARTHRITIS, RHEUMATOID; FEMALE; HUMANS; PARKINSON DISEASE, SECONDARY; TUMOR NECROSIS FACTOR-ALPHA;

EID: 0042997024     PISSN: 14620324     EISSN: None     Source Type: Journal    
DOI: 10.1093/rheumatology/keg160     Document Type: Letter

References (8)

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8. Gayle DA, Ling Z, Tong C, Landers T, Lipton JW, Carvey PM. Lipopolysaccharide (LPS)-induced dopamine cell loss in culture: roles of tumor necrosis factor-alpha, interleukin-1 beta, and nitric oxide. Brain Res Dev Brain Res 2002;133:27-35.