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Volumn 9, Issue 4, 2003, Pages

Persistence of HIV-1 drug resistance mutations and emergence during antiretroviral treatment interruption: Considerations from a clinical case

Author keywords

Drug resistance; HIV; Persistence; Proviral DNA

Indexed keywords

ANTIRETROVIRUS AGENT; INDINAVIR; PROTEINASE INHIBITOR; RNA DIRECTED DNA POLYMERASE INHIBITOR; ZALCITABINE; ZIDOVUDINE;

EID: 0038559640     PISSN: 12341010     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article
Times cited : (4)

References (12)
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  • 2
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  • 3
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  • 4
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    • Garcia F, Vidal C, Plana M: Residual low-level viral replication could explain discrepancies between viral load and CD4+ cell response in human immunodeficiency virus-infected patients receiving antiretroviral therapy. Clin Infect Dis, 2000; 30: 392-394
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  • 6
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    • Productive infection maintains a dynamic steady-state of residual viraemia in HIV-1 infected persons treated with suppressive antiretroviral therapy for 5 years
    • Havlir D, Strain M, Clerici M et al: Productive infection maintains a dynamic steady-state of residual viraemia in HIV-1 infected persons treated with suppressive antiretroviral therapy for 5 years. Antiviral Ther, 2002; 7: 41
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  • 7
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    • Drug resistance at low viremia in HIV-infected patients with antiretroviral combination therapy
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  • 8
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    • Detection of resistance mutations in patients receiving a first line protease inhibitor containing regimen with undetectable plasma viral load
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  • 9
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    • Accessed 3rd September
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.