Heteromerization of Kir2.x potassium channels contributes to the phenotype of Andersen's syndrome

Proceedings of the National Academy of Sciences of the United States of America

Volumn 99, Issue 11, 2002, Pages 7774-7779

  Preisig Müller, Regina ^a   Schlichthörl, Günter ^a   Goerge, Tobias ^b   Heinen, Steffen ^b   Brüggemann, Andrea ^b   Rajan, Sindhu ^a   Derst, Christian ^a   Veh, Rüdiger W ^c   Daut, Jürgen ^a  

^a PHILIPPS UNIVERSITY MARBURG   (Germany)

^b SANOFI AVENTIS DEUTSCHLAND GMBH   (Germany)

^c CHARITÉ UNIVERSITÄTSMEDIZIN BERLIN   (Germany)

Author keywords

[No Author keywords available]

Indexed keywords

BARIUM; MUTANT PROTEIN; POTASSIUM CHANNEL; PROTEIN SUBUNIT; TETRAMER;

AMINO TERMINAL SEQUENCE; ANIMAL CELL; ARTICLE; AUTOSOMAL DOMINANT DISORDER; CARBOXY TERMINAL SEQUENCE; GENE EXPRESSION; GENE MUTATION; GLYCOGEN STORAGE DISEASE TYPE 4; GUINEA PIG; HEART MUSCLE FIBER MEMBRANE; HUMAN; HUMAN CELL; IMMUNOPRECIPITATION; MEMBRANE CURRENT; MOLECULAR INTERACTION; NONHUMAN; NUCLEOTIDE SEQUENCE; OLIGOMERIZATION; PHENOTYPE; PLEIOTROPY; POTASSIUM CHANNEL KIR2 X; PRIORITY JOURNAL; PROTEIN DOMAIN; TWO HYBRID SYSTEM; XENOPUS;

AMINO ACID SUBSTITUTION; ANIMALS; CELL LINE; GUINEA PIGS; HUMANS; LONG QT SYNDROME; MODELS, MOLECULAR; MUTAGENESIS, INSERTIONAL; PARALYSES, FAMILIAL PERIODIC; PHENOTYPE; POTASSIUM CHANNELS, INWARDLY RECTIFYING; PROTEIN CONFORMATION; PROTEIN SUBUNITS; TRANSFECTION;

ANIMALIA; CAVIA PORCELLUS; SUS SCROFA;

EID: 0037188493     PISSN: 00278424     EISSN: None     Source Type: Journal    
DOI: 10.1073/pnas.102609499     Document Type: Article

References (41)