Nitric oxide inhibition in cirrhosis and ascites [10]

American Journal of Gastroenterology

Volumn 98, Issue 7, 2003, Pages 1666-1667

  Cardenas, Andres ^a  

^a HARVARD MEDICAL SCHOOL   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

N(G) METHYLARGININE; N(G) NITROARGININE METHYL ESTER; NITRIC OXIDE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ASCITES; CONTINUOUS INFUSION; DRUG EFFECT; DRUG INFUSION; DRUG MECHANISM; GLOMERULUS FILTRATION RATE; HEMODYNAMICS; KIDNEY BLOOD FLOW; KIDNEY FUNCTION; LETTER; LIVER BLOOD FLOW; LIVER CIRRHOSIS; NONHUMAN; PATHOGENESIS; PRIORITY JOURNAL; RAT; SODIUM EXCRETION;

EID: 0037780481     PISSN: 00029270     EISSN: None     Source Type: Journal    
DOI: 10.1111/j.1572-0241.2003.07559.x     Document Type: Letter

References (5)

1. Thiesson HC, Skott O, Jespersen B, et al. Nitric oxide synthase inhibition does not improve renal function in cirrhotic patients with ascites. Am J Gastroenterol 2003;98:180-6.
2. Martin PY, Gines P, Schrier RW. Nitric oxide as a mediator of hemodynamic abnormalities and sodium and water retention in cirrhosis. N Engl J Med 1998;20:339:533-41.
3. Martin PY, Ohara M, Gines P, et al. Nitric oxide synthase (NOS) inhibition for one week improves renal sodium and water excretion in cirrhotic rats with ascites. J Clin Invest 1998;101:235-42.
4. La Villa G, Barletta E, Pantaleo P, et al. Hemodynamic, renal, and endocrine effects of acute inhibition of nitric oxide synthase in compensated cirrhosis. Hepatology 2001;34:19-27.
5. Spahr L, Martin PY, Giostra E, et al. Acute effects of nitric oxide synthase (NOS) inhibition on systemic, hepatic and renal hemodynamics in patients with cirrhosis and ascites. J Invest Med 2002;50:116-24.