Cardiomyocyte apoptosis is associated with increased wall stress in chronic failing left ventricle

European Heart Journal

Volumn 24, Issue 8, 2003, Pages 742-751

  Jiang, L ^a,b   Huang, Y ^a   Hunyor, S ^a   Dos Remedios, C G ^b  

^a ROYAL NORTH SHORE HOSPITAL   (Australia)

^b UNIVERSITY OF SYDNEY   (Australia)

Author keywords

Apoptosis; Caspase; Chronic heart failure; Fas Fas ligand; Intercalated disc; Wall stress

Indexed keywords

CASPASE 3; CASPASE 8; FAS ANTIGEN; FAS LIGAND; PROTEIN; PROTEIN P20; PROTEINASE; UNCLASSIFIED DRUG;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CONTROLLED STUDY; CORONARY ARTERY OBSTRUCTION; DIASTOLIC BLOOD PRESSURE; DISEASE ASSOCIATION; ENZYME ACTIVATION; HEART DILATATION; HEART LEFT VENTRICLE FAILURE; HEART MUSCLE CELL; HEART STRESS; HEMODYNAMICS; IMMUNOHISTOCHEMISTRY; MICROEMBOLISM; MOLECULAR INTERACTION; NICK END LABELING; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN LOCALIZATION; SHEEP; WESTERN BLOTTING;

ANIMALS; ANTIGENS, CD95; APOPTOSIS; BLOTTING, WESTERN; CARDIAC OUTPUT, LOW; CASPASE 3; CASPASE 8; CASPASE 9; CASPASES; CHRONIC DISEASE; FAS LIGAND PROTEIN; IMMUNOHISTOCHEMISTRY; IN SITU NICK-END LABELING; MEMBRANE GLYCOPROTEINS; MYOCYTES, CARDIAC; SHEEP; STRESS; VENTRICULAR DYSFUNCTION, LEFT;

EID: 0037393472     PISSN: 0195668X     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0195-668X(02)00655-3     Document Type: Article

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