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Volumn 5, Issue 2, 2003, Pages 222-223
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Molecular mechanisms of nickel carcinogenesis: Gene silencing by nickel delivery to the nucleus and gene activation/inactivation by nickel-induced cell signaling
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Author keywords
[No Author keywords available]
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Indexed keywords
CELL SURFACE RECEPTOR;
HISTONE;
NICKEL;
ACETYLATION;
ANIMAL CELL;
CANCER CELL;
CANCER RISK;
CARCINOGENESIS;
CELL LEVEL;
CELL NUCLEUS;
CELL SURVIVAL;
CELL TRANSFORMATION;
CONCENTRATION (PARAMETERS);
CONFERENCE PAPER;
DNA METHYLATION;
GENE ACTIVATION;
GENE EXPRESSION;
GENE INACTIVATION;
GENE INDUCTION;
GENE SILENCING;
HETEROCHROMATIN;
METASTASIS;
MOLECULAR INTERACTION;
NONHUMAN;
PRIORITY JOURNAL;
SIGNAL TRANSDUCTION;
SOLUBILITY;
TUMOR SUPPRESSOR GENE;
CALCIUM SIGNALING;
CELL TRANSFORMATION, NEOPLASTIC;
CHROMATIN;
DISEASE PROGRESSION;
DNA DAMAGE;
DNA METHYLATION;
DNA-BINDING PROTEINS;
HUMANS;
HYPOXIA-INDUCIBLE FACTOR 1;
HYPOXIA-INDUCIBLE FACTOR 1, ALPHA SUBUNIT;
NEOPLASMS;
NICKEL;
NUCLEAR PROTEINS;
RISK ASSESSMENT;
SIGNAL TRANSDUCTION;
TRANSCRIPTION FACTORS;
ANIMALIA;
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EID: 0037392103
PISSN: 14640325
EISSN: None
Source Type: Journal
DOI: 10.1039/b210260a Document Type: Conference Paper |
Times cited : (53)
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References (24)
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