Ultrafine carbon black particles enhance respiratory syncytial virus-induced airway reactivity, pulmonary inflammation, and chemokine expression

Toxicological Sciences

Volumn 72, Issue 2, 2003, Pages 339-346

  Lambert, Amy L ^a   Mangum, James B ^a   DeLorme, Michael P ^a   Everitt, Jeffrey I ^a  

^a CIIT CENTERS FOR HEALTH RESEARCH   (United States)

Author keywords

Airway hyperresponsiveness; Chemokine; Cytokine; Laser capture microdissection; Mouse; RSV; Ultrafine particles

Indexed keywords

CARBON; CHEMOKINE; MACROPHAGE INFLAMMATORY PROTEIN 1ALPHA; MESSENGER RNA; METHACHOLINE; MONOCYTE CHEMOTACTIC PROTEIN 1; RANTES;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; BRONCHUS HYPERREACTIVITY; CONTROLLED STUDY; CULTURE MEDIUM; FEMALE; IMMUNOREACTIVITY; IMMUNOREGULATION; LUNG ALVEOLUS EPITHELIUM; LUNG CLEARANCE; LUNG INJURY; LUNG LAVAGE; MOUSE; MOUSE STRAIN; NEUTROPHIL; NONHUMAN; PARTICLE SIZE; PARTICULATE MATTER; PATHOGENICITY; PROTEIN EXPRESSION; PROTEIN SECRETION; REGULATORY MECHANISM; RESPIRATORY SYNCYTIAL PNEUMOVIRUS; RNA VIRUS INFECTION; T LYMPHOCYTE ACTIVATION;

ANIMALS; BRONCHI; BRONCHIAL HYPERREACTIVITY; BRONCHOALVEOLAR LAVAGE FLUID; CARBON; CELL COUNT; CHEMOKINES; FEMALE; INTUBATION, INTRATRACHEAL; MACROPHAGE INFLAMMATORY PROTEIN-1; MICE; MICE, INBRED BALB C; PARTICLE SIZE; PNEUMONIA, VIRAL; RESPIRATORY SYNCYTIAL VIRUS INFECTIONS; RESPIRATORY SYNCYTIAL VIRUSES; RNA, MESSENGER; SPECIFIC PATHOGEN-FREE ORGANISMS;

ANIMALIA; BRONCHUS; PNEUMOVIRUS; RESPIRATORY SYNCYTIAL VIRUS; RICE STRIPE VIRUS; RNA VIRUSES;

EID: 0037389340     PISSN: 10966080     EISSN: None     Source Type: Journal    
DOI: 10.1093/toxsci/kfg032     Document Type: Article

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