C57BLack/BOX? The importance of exact mouse strain nomenclature

Trends in Genetics

Volumn 19, Issue 4, 2003, Pages 183-184

  Wotjak, Carsten T ^a  

^a MAX PLANCK INSTITUTE OF PSYCHIATRY   (Germany)

Author keywords

[No Author keywords available]

Indexed keywords

ALPHA SYNUCLEIN; PRESYNAPTIC TELENCEPHALIC PROTEIN; PROTEIN DERIVATIVE; UNCLASSIFIED DRUG;

ALZHEIMER DISEASE; ANIMAL BEHAVIOR; CHROMOSOME DELETION; CONTROLLED STUDY; DNA MODIFICATION; GENETIC ASSOCIATION; LETTER; MOUSE; MOUSE STRAIN; NOMENCLATURE; NONHUMAN; PARKINSON DISEASE; PHENOTYPE; PHYSIOLOGY; PRIORITY JOURNAL; SINGLE NUCLEOTIDE POLYMORPHISM; TRANSGENIC ANIMAL;

ANIMALIA;

EID: 0037384725     PISSN: 01689525     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0168-9525(02)00049-5     Document Type: Letter

References (7)

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3. Wade C.M., et al. The mosaic structure of variation in the mouse genome. Nature. 420:2002;574-578.
4. Specht C.G., Schoepfer R. Deletion of the alpha-synuclein locus in a subpopulation of C57BL /6J inbred mice. BMC Neurosci. 2:2001;11.
5. Clayton D.F., George J.M. The synucleins: a family of proteins involved in synaptic function, plasticity, neurodegeneration and disease. Trends Neurosci. 21:1998;249-254.
6. Masliah E., et al. Dopaminergic loss and inclusion body formation in alpha-synuclein mice: implications for neurodegenerative disorders. Science. 287:2000;1265-1269.
7. Chen P.E., et al. Spatial learning is unimpaired in mice containing a deletion of the alpha-synuclein locus. Eur. J. Neurosci. 16:2002;154-158.