C-terminal fragments of amyloid-β peptide cause cholinergic axonal degeneration by a toxic effect rather than by physical injury in the nondemented human brain

Neurochemical Research

Volumn 28, Issue 3-4, 2003, Pages 493-498

  Kasa, Peter ^a   Papp, Henrietta ^a   Zombori, Janos ^b   Mayer, Peter ^b   Checler, Frederic ^c  

^a UNIVERSITY OF SZEGED   (Hungary)

^b Elisabeth Hospital   (Hungary)

^c INSTITUT DE PHARMACOLOGIE MOLÉCULAIRE ET CELLULAIRE   (France)

Author keywords

Acetylcholinesterase; Aging; Amyloid ; Chemical toxicity; Dystrophic neurite; Physical injury

Indexed keywords

ACETYLCHOLINESTERASE; AMYLOID BETA PROTEIN;

AGED; AMINO TERMINAL SEQUENCE; ARTICLE; BRAIN; CARBOXY TERMINAL SEQUENCE; CLINICAL ARTICLE; CONTROLLED STUDY; DEMENTIA; DYSTROPHY; ENTORHINAL CORTEX; HIPPOCAMPUS; HISTOCHEMISTRY; HUMAN; HUMAN TISSUE; IMMUNOHISTOCHEMISTRY; NERVE FIBER; NERVE FIBER DEGENERATION; NEURITE; NEUROTOXICITY; NORMAL HUMAN; PHYSICAL DISEASE; PRIORITY JOURNAL; SENILE PLAQUE;

ACETYLCHOLINESTERASE; AGED; AGED, 80 AND OVER; AMYLOID BETA-PROTEIN; AXONS; BRAIN DISEASES; CHOLINERGIC FIBERS; HUMANS; NERVE DEGENERATION; NEURITES; PEPTIDE FRAGMENTS; SENILE PLAQUES; TAU PROTEINS; TISSUE DISTRIBUTION;

EID: 0037375473     PISSN: 03643190     EISSN: None     Source Type: Journal    
DOI: 10.1023/A:1022813104905     Document Type: Article

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