Interleukin-13 induces surfactant function abnormality in the murine lung

Chest

Volumn 123, Issue 3 SUPPL., 2003, Pages 375S-376S

  Zhu, Zhou ^a   Enhorning, Goran ^b   Zheng, Tao ^a   Chen, Qingsheng ^a   Chen, Ning Yuan ^a   Homer, Robert ^a   Elias, Jack A ^a  

^a YALE UNIVERSITY SCHOOL OF MEDICINE   (United States)

^b UNIVERSITY AT BUFFALO   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ALLERGEN; COMPLEMENTARY DNA; GENE PRODUCT; INTERLEUKIN 5; LUNG SURFACTANT; MESSENGER RNA; PHOSPHATIDYLCHOLINE; PHOSPHATIDYLGLYCEROL; PHOSPHOLIPASE A2; RECOMBINANT ENZYME;

ALLERGIC ASTHMA; CELL FUNCTION; CELL INFILTRATION; CONFERENCE PAPER; CONTROLLED STUDY; DNA SEQUENCE; ENZYME RELEASE; ENZYME SPECIFICITY; EOSINOPHIL; EPITHELIUM CELL; GENE EXPRESSION; HUMAN; HUMAN CELL; HYDROLYSIS; IN SITU HYBRIDIZATION; IN VITRO STUDY; LUNG LAVAGE; MOLECULAR CLONING; PRIORITY JOURNAL; PROVOCATION TEST; RESPIRATORY EPITHELIUM; REVERSE TRANSCRIPTION POLYMERASE CHAIN REACTION; RNA ANALYSIS; RNA ISOLATION;

EID: 0037335831     PISSN: 00123692     EISSN: None     Source Type: Journal    
DOI: 10.1378/chest.123.3_suppl.375S     Document Type: Conference Paper

References (2)

1. Zhu Z, Homer RJ, Wang Z, et al. Pulmonary expression of interleukin-13 causes inflammation, mucus hypersecretion, subepithelial fibrosis, physiologic abnormalities, and eotaxin production. J Clin Invest 1999; 103:779-788
2. Homer RJ, Zheng T, Chupp G, et al. Pulmonary type II cell hypertrophy and pulmonary lipoproteinosis are features of chronic IL-13 exposure. Am J Physiol 2002; 283:L52-L59