-
1
-
-
0030889389
-
Interaction of tumor and host cells with adhesion and extracellular matrix molecules in the development of multiple myeloma
-
Teoh G, Anderson C: Interaction of tumor and host cells with adhesion and extracellular matrix molecules in the development of multiple myeloma. Hematol Oncol Clin North Am 11:27-42, 1997
-
(1997)
Hematol Oncol Clin North Am
, vol.11
, pp. 27-42
-
-
Teoh, G.1
Anderson, C.2
-
2
-
-
0030041147
-
Multiple myeloma cell adhesiom-induced interleukin-6 expression in bone marrow stromal cells involves activation of NF-kappa B
-
Chauhan D, Uchiyama H, Akbarali Y, et al: Multiple myeloma cell adhesiom-induced interleukin-6 expression in bone marrow stromal cells involves activation of NF-kappa B. Blood 87:1104-1112, 1996
-
(1996)
Blood
, vol.87
, pp. 1104-1112
-
-
Chauhan, D.1
Uchiyama, H.2
Akbarali, Y.3
-
3
-
-
0035958517
-
The role of tumor necrosis factor in the pathophysiology of human multiple myeloma: Therapeutic applications
-
Hideshima T, Chauhan D, Schlossman R, et al: The role of tumor necrosis factor in the pathophysiology of human multiple myeloma: therapeutic applications. Oncogene 20:4519-4527, 2001
-
(2001)
Oncogene
, vol.20
, pp. 4519-4527
-
-
Hideshima, T.1
Chauhan, D.2
Schlossman, R.3
-
4
-
-
0030026437
-
1: Differential effects on multiple myeloma versus normal B cells
-
1: Differential effects on multiple myeloma versus normal B cells. Blood 87:1928-1938, 1996
-
(1996)
Blood
, vol.87
, pp. 1928-1938
-
-
Urashima, M.1
Ogata, A.2
Chauhan, D.3
-
5
-
-
0035880256
-
Vascular endothelial growth factor triggers signaling cascades mediating multiple myeloma cell growth and migration
-
Podar K, Tai YT, Davies FE, et al: Vascular endothelial growth factor triggers signaling cascades mediating multiple myeloma cell growth and migration. Blood 98:428-435, 2001
-
(2001)
Blood
, vol.98
, pp. 428-435
-
-
Podar, K.1
Tai, Y.T.2
Davies, F.E.3
-
6
-
-
0031225483
-
Interleukin-6 triggers cell growth via the ras-dependent mitogen-activated protein kinase cascade
-
Ogata A, Chauhan D, Teoh G, et al: Interleukin-6 triggers cell growth via the ras-dependent mitogen-activated protein kinase cascade. J Immunol 159:2212-2221, 1997
-
(1997)
J Immunol
, vol.159
, pp. 2212-2221
-
-
Ogata, A.1
Chauhan, D.2
Teoh, G.3
-
7
-
-
0034331194
-
Thalidomide and its analogs overcome drug resistance of human multiple myeloma cells to conventional therapy
-
Hideshima T, Chauhan D, Shima Y, et al: Thalidomide and its analogs overcome drug resistance of human multiple myeloma cells to conventional therapy. Blood 96:2943-2950, 2000
-
(2000)
Blood
, vol.96
, pp. 2943-2950
-
-
Hideshima, T.1
Chauhan, D.2
Shima, Y.3
-
8
-
-
0035195019
-
Adherence of multiple myeloma cells to bone marrow stromal cells upregulates vascular endothelial growth factor secretion: Therapeutic applications
-
Gupta D, Treon SP, Shima Y, et al: Adherence of multiple myeloma cells to bone marrow stromal cells upregulates vascular endothelial growth factor secretion: Therapeutic applications. Leukemia 15:1950-1961, 2001
-
(2001)
Leukemia
, vol.15
, pp. 1950-1961
-
-
Gupta, D.1
Treon, S.P.2
Shima, Y.3
-
9
-
-
0012753815
-
Thalidomide and its immunomodulatory analogs inhibit human multiple myeloma cell growth and angiogenesis in vivo
-
in press
-
Lentzsch S, LeBlanc R, Podar K, et al: Thalidomide and its immunomodulatory analogs inhibit human multiple myeloma cell growth and angiogenesis in vivo. Leukemia (in press)
-
Leukemia
-
-
Lentzsch, S.1
LeBlanc, R.2
Podar, K.3
-
10
-
-
0035412366
-
Thalidomide and immunomodulatory derivatives augment natural killer cell cytotoxicity in multiple myeloma
-
Davies FE, Raje N, Hideshima T, et al: Thalidomide and immunomodulatory derivatives augment natural killer cell cytotoxicity in multiple myeloma. Blood 98:210-216, 2001
-
(2001)
Blood
, vol.98
, pp. 210-216
-
-
Davies, F.E.1
Raje, N.2
Hideshima, T.3
-
11
-
-
0034999619
-
Tumor cell expression of CD59 is associated with resistance to CD20 serotherapy in B-cell malignancies
-
Treon SP, Mitsiades C, Mitsiades N, et al: Tumor cell expression of CD59 is associated with resistance to CD20 serotherapy in B-cell malignancies. J Immunother 24:263-271, 2001
-
(2001)
J Immunother
, vol.24
, pp. 263-271
-
-
Treon, S.P.1
Mitsiades, C.2
Mitsiades, N.3
-
12
-
-
0012778205
-
A phase 1 study of oral CC5013, an immunomodulatory thalidomide (Thal) derivative, in patients with relapsed and refractory multiple myeloma (MM)
-
December, Orlando, FL (abstr 3225)
-
Richardson PG, Schlossman RL, Hideshima T: A phase 1 study of oral CC5013, an immunomodulatory thalidomide (Thal) derivative, in patients with relapsed and refractory multiple myeloma (MM). Presented at the American Society of Hematology 43rd Annual Meeting, December 2001, Orlando, FL (abstr 3225)
-
(2001)
American Society of Hematology 43rd Annual Meeting
-
-
Richardson, P.G.1
Schlossman, R.L.2
Hideshima, T.3
-
13
-
-
0031985198
-
Multiple myeloma: Increasing evidence for a multistep transformation process
-
Hallek M, Bergsagel PL, Anderson KC: Multiple myeloma: increasing evidence for a multistep transformation process. Blood 91:3-21, 1998
-
(1998)
Blood
, vol.91
, pp. 3-21
-
-
Hallek, M.1
Bergsagel, P.L.2
Anderson, K.C.3
-
14
-
-
0035300479
-
The proteasome inhibitor PS-341 inhibits growth, induces apoptosis, and overcomes drug resistance in human multiple myeloma cells
-
Hideshima T, Richardson P, Chauhan D, et al: The proteasome inhibitor PS-341 inhibits growth, induces apoptosis, and overcomes drug resistance in human multiple myeloma cells. Cancer Res 61:3071-3076, 2001
-
(2001)
Cancer Res
, vol.61
, pp. 3071-3076
-
-
Hideshima, T.1
Richardson, P.2
Chauhan, D.3
-
15
-
-
0012778206
-
Phase II study of PS-341, a novel proteasome inhibitor, alone or in combination with dexamethasone in patients with multiple myeloma who have relapsed following front-line therapy and are refractory to their most recent therapy
-
December, Orlando, FL (abstr 3223)
-
Richardson P: Phase II study of PS-341, a novel proteasome inhibitor, alone or in combination with dexamethasone in patients with multiple myeloma who have relapsed following front-line therapy and are refractory to their most recent therapy. Presented at the American Society of Hematology 43rd Annual Meeting, December 2001, Orlando, FL (abstr 3223)
-
(2001)
American Society of Hematology 43rd Annual Meeting
-
-
Richardson, P.1
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