SCOPUS 정보 검색 플랫폼

Volumn 21, Issue 38, 2002, Pages 5844-5851

DNA damage, death receptor activation and reactive oxygen species contribute to ultraviolet radiation-induced apoptosis in an essential and independent way

(4) Kulms, Dagmar a Zeise, Elke a Pöppelmann, Birgit a Schwarz, Thomas a

a UNIVERSITY OF MÜNSTER (Germany)

Author keywords

Apoptosis; Cytochrome c; DNA damage; Reactive oxygen species; UV radiation

Indexed keywords

CASPASE 8; CASPASE INHIBITOR; CELL DNA; CYTOCHROME C; FAS ANTIGEN; HYDROGEN PEROXIDE; OLIGOPEPTIDE; PYRROLIDINE DITHIOCARBAMATE; REACTIVE OXYGEN METABOLITE;

APOPTOSIS; ARTICLE; CELL DEATH; CONTROLLED STUDY; DNA DAMAGE; HELA CELL; HUMAN; HUMAN CELL; OXIDATIVE STRESS; PRIORITY JOURNAL; SIGNAL TRANSDUCTION; ULTRAVIOLET B RADIATION; ULTRAVIOLET IRRADIATION;

ANTIGENS, CD95; APOPTOSIS; CELL DEATH; CYSTEINE PROTEINASE INHIBITORS; CYTOCHROME C GROUP; DNA DAMAGE; DNA REPAIR; HELA CELLS; HUMANS; HYDROGEN PEROXIDE; OLIGOPEPTIDES; PROLINE; REACTIVE OXYGEN SPECIES; SIGNAL TRANSDUCTION; THIOCARBAMATES; ULTRAVIOLET RAYS;

EID: 0037194610 PISSN: 09509232 EISSN: None Source Type: Journal
DOI: 10.1038/sj.onc.1205743 Document Type: Article

Times cited : (228)

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