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Volumn 39, Issue 2, 2002, Pages 351-358
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Addition of spironolactone to angiotensin-converting enzyme inhibition in heart failure improves endothelial vasomotor dysfunction: Role of vascular superoxide anion formation and endothelial nitric oxide synthase expression
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Author keywords
[No Author keywords available]
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Indexed keywords
DIPEPTIDYL CARBOXYPEPTIDASE INHIBITOR;
NITRIC OXIDE;
NITRIC OXIDE SYNTHASE;
PLACEBO;
SPIRONOLACTONE;
TRANDOLAPRIL;
ANIMAL MODEL;
ARTICLE;
CONTROLLED STUDY;
HEART FAILURE;
HEART INFARCTION SIZE;
HEART MUSCLE PERFUSION;
HEART VENTRICLE REMODELING;
HEMODYNAMIC MONITORING;
MALE;
MODULATION;
NONHUMAN;
PRIORITY JOURNAL;
PROTEIN EXPRESSION;
RAT;
TREATMENT OUTCOME;
VASCULAR RING;
VASODILATATION;
ALDOSTERONE ANTAGONISTS;
ANGIOTENSIN-CONVERTING ENZYME INHIBITORS;
ANIMALS;
DRUG THERAPY, COMBINATION;
ENDOTHELIUM, VASCULAR;
HEART FAILURE, CONGESTIVE;
INDOLES;
MALE;
NITRIC OXIDE SYNTHASE;
NITRIC OXIDE SYNTHASE TYPE III;
RATS;
RATS, WISTAR;
SPIRONOLACTONE;
SUPEROXIDES;
VASODILATION;
VENTRICULAR REMODELING;
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EID: 0037116558
PISSN: 07351097
EISSN: None
Source Type: Journal
DOI: 10.1016/S0735-1097(01)01729-6 Document Type: Article |
Times cited : (141)
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References (36)
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