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Volumn 403, Issue 2, 2002, Pages 189-201
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The silencing mediator of retinoic acid and thyroid hormone receptors can interact with the aryl hydrocarbon (Ah) receptor but fails to repress Ah receptor-dependent gene expression
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Author keywords
ARNT; Aryl hydrocarbon receptor; Dioxin; SMRT; TCDD; Transcriptional regulation
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Indexed keywords
2,3,7,8 TETRACHLORODIBENZO PARA DIOXIN;
AROMATIC HYDROCARBON RECEPTOR;
CYTOCHROME P450 1A1;
DIMER;
GLUTATHIONE TRANSFERASE;
RETINOIC ACID;
THYROID HORMONE RECEPTOR;
TRANSCRIPTION FACTOR;
ARTICLE;
GENE EXPRESSION;
GENE REPRESSION;
GENE SILENCING;
GENETIC TRANSCRIPTION;
GENETIC TRANSFECTION;
MOLECULAR RECOGNITION;
NONHUMAN;
PRIORITY JOURNAL;
PROTEIN DOMAIN;
PROTEIN PROTEIN INTERACTION;
REPORTER GENE;
SIGNAL TRANSDUCTION;
TRANSCRIPTION REGULATION;
ANIMALS;
ARYL HYDROCARBON RECEPTOR NUCLEAR TRANSLOCATOR;
BASE SEQUENCE;
BREAST NEOPLASMS;
CYTOCHROME P-450 CYP1A1;
DNA-BINDING PROTEINS;
GENE EXPRESSION REGULATION;
GLUTATHIONE TRANSFERASE;
HUMANS;
MICE;
MOLECULAR SEQUENCE DATA;
PROMOTER REGIONS (GENETICS);
RECEPTORS, ARYL HYDROCARBON;
RECOMBINANT PROTEINS;
REPRESSOR PROTEINS;
RESPONSE ELEMENTS;
SPECIES SPECIFICITY;
TETRACHLORODIBENZODIOXIN;
TRANSCRIPTION FACTORS;
TUMOR CELLS, CULTURED;
MAMMALIA;
MINK CELL FOCUS-FORMING VIRUS;
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EID: 0037099253
PISSN: 00039861
EISSN: None
Source Type: Journal
DOI: 10.1016/S0003-9861(02)00233-3 Document Type: Article |
Times cited : (47)
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References (57)
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