Pathogenesis of lupus

Arthritis Care and Research

Volumn 47, Issue 3, 2002, Pages 343-345

  Oelke, Kurt ^a   Richardson, Bruce ^a,b  

^a UNIVERSITY OF MICHIGAN   (United States)

^b Geriatrics Center   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CYCLIC AMP DEPENDENT PROTEIN KINASE; FC RECEPTOR;

DNA METHYLATION; ENZYME DEFICIENCY; GENETIC PREDISPOSITION; HUMAN; NUCLEOSOME; PATHOGENESIS; RECEPTOR INTRINSIC ACTIVITY; SHORT SURVEY; SYSTEMIC LUPUS ERYTHEMATOSUS;

EID: 0037097822     PISSN: 21514658     EISSN: None     Source Type: Journal    
DOI: 10.1002/art.10380     Document Type: Short Survey

References (17)

1. Genome scan of human systemic lupus erythematosus by regression modeling: Evidence of linkage and epistasis at 4p16-15.2
2. Genetics and systemic lupus erythematosus
3. The key role of nucleosomes in lupus
4. Molecular aberrations in human systemic lupus erythematosus
5. Evidence for impaired T cell DNA methylation in systemic lupus erythematosus and rheumatoid arthritis
6. Treating activated CD4+ T cells with either of two distinct DNA methyltransferase inhibitors, 5-azacytidine or procainamide, is sufficient to cause a lupus-like disease in syngeneic mice
7. The Fcγreceptor IIIA-158F allele is a major risk factor for the development of lupus nephritis among Caucasians but not non-Caucasians
8. Nucleosomes are major T and B cell autoantigens in systemic lupus erythematosus
9. The 1982 revised criteria for the classification of systemic lupus erythematosus
10. Systemic exposure to irradiated apoptotic cells induces autoantibody production
11. Nucleosome: A major immunogen for pathogenic autoantibody-inducing T cells of lupus
12. High prevalence of T cell type I protein kinase A deficiency in systemic lupus erythematosus
13. mRNA mutations of type I protein kinase A regulatory subunit alpha in T lymphocytes of a subject with systemic lupus erythematosus
14. Protein kinase a RI beta subunit deficiency in lupus T lymphocytes: Bypassing a block in RI beta translation reconstitutes protein kinase A activity and augments IL-2 production
15. Decreased ras-mitogen-activated protein kinase signaling may cause DNA hypomethylation in T lymphocytes from lupus patients
16. Mechanisms of drug-induced lupus. II. T cells overexpressing lymphocyte function-associated antigen 1 become autoreactive and cause a lupus-like disease in syngeneic mice
17. Phenotypic and functional similarities between 5-azacytidine-treated T cells and a T cell subset in patients with active systemic lupus erythematosus