Expression of the complement C3a and C5a receptors after permanent focal ischemia: An alternative interpretation

GLIA

Volumn 38, Issue 2, 2002, Pages 169-173

  Barnum, Scott R ^a   Ames, Robert S ^b   Maycox, Peter R ^c   Hadingham, Sarah J ^c   Meakin, Jackie ^c   Harrison, David ^c   Parsons, Andrew A ^c  

^a University of Alabama at Birmingham   (United States)

^b GLAXOSMITHKLINE   (United States)

^c GLAXOSMITHKLINE   (United Kingdom)

Author keywords

Inflammation; Murine; Neurons; Stroke

Indexed keywords

COMPLEMENT C3A RECEPTOR; COMPLEMENT COMPONENT C3A; COMPLEMENT COMPONENT C5A; COMPLEMENT COMPONENT C5A RECEPTOR; COMPLEMENT RECEPTOR; LEUKOCYTE ANTIGEN; MEMBRANE PROTEIN; MESSENGER RNA;

ANIMAL; ARTICLE; CEREBRAL ARTERY DISEASE; ENCEPHALITIS; GENE EXPRESSION; GENETICS; IMMUNOLOGY; MALE; MOUSE; MOUSE STRAIN; PATHOPHYSIOLOGY; RAT; SPRAGUE DAWLEY RAT;

ANIMALS; ANTIGENS, CD; COMPLEMENT C3A; COMPLEMENT C5A; ENCEPHALITIS; GENE EXPRESSION; INFARCTION, MIDDLE CEREBRAL ARTERY; MALE; MEMBRANE PROTEINS; MICE; MICE, INBRED STRAINS; RATS; RATS, SPRAGUE-DAWLEY; RECEPTOR, ANAPHYLATOXIN C5A; RECEPTORS, COMPLEMENT; RNA, MESSENGER;

EID: 0037089099     PISSN: 08941491     EISSN: None     Source Type: Journal    
DOI: 10.1002/glia.10069     Document Type: Article

References (13)

1. Procedural and strain-related variables significantly affect outcome in a murine model of focal cerebral ischemia
2. Receptor for the C3a anaphylatoxin is expressed by neurons and glial cells
3. Complement components of the innate immune system in health and disease in the CNS
4. The use of quantitative RT-PCR to measure mRNA expression in a rat model of focal ischemia - Caspase-3 as a case study
5. Neuronal protection in stroke by an sLex-glycosylated complement inhibitory protein
6. Reversible middle cerebral artery occlusion without craniectomy in rats
7. The use of TaqMan RT-PCR assays for semi-quantitative analysis of gene expression in CNS tissues and disease models
8. Complement anaphylatoxin C5a neuroprotects through mitogen-activated protein kinase-dependent inhibition of caspase-3
9. Complement anaphylatoxin receptors on neurons: New tricks for old receptors?
10. Complement C1q is dramatically up-regulated in brain microglia in response to transient global cerebral ischemia
11. The role of the complement system in traumatic brain injury
12. Hypoxia-induced expression of c1q, a subcomponent of the complement system, in cultured rat PC12 cells
13. Expression of receptors for complement anaphylatoxins C3a and C5a following permanent focal cerebral ischemia in the mouse