Non-TH1 cytokines are augmented systematically early in Guillain-Barré syndrome

Neurology

Volumn 58, Issue 3, 2002, Pages 476-478

  Press, Rayomand ^a   Ozenci, V ^a   Kouwenhoven, M ^a   Link, H ^a  

^a KAROLINSKA UNIVERSITY HOSPITAL   (Sweden)

Author keywords

[No Author keywords available]

Indexed keywords

CYTOKINE; IMMUNOGLOBULIN; INTERLEUKIN 10; INTERLEUKIN 12; INTERLEUKIN 6; TUMOR NECROSIS FACTOR ALPHA;

ARTICLE; CLINICAL ARTICLE; CONTROLLED STUDY; CYTOKINE RELEASE; DISEASE ACTIVITY; DISEASE ASSOCIATION; DISEASE COURSE; ENZYME IMMUNOASSAY; GUILLAIN BARRE SYNDROME; HUMAN; HUMAN CELL; MONONUCLEAR CELL; PRIORITY JOURNAL; TH1 CELL;

EID: 0037065844     PISSN: 00283878     EISSN: None     Source Type: Journal    
DOI: 10.1212/WNL.58.3.476     Document Type: Article

References (10)

1. Pathogenesis of Guillain-Barré syndrome
2. Elevated serum levels of IFN-gamma, IL-4 and TNF-alpha/unelevated serum levels of IL-10 in patients with demyelinating diseases during the acute stage
3. Cytokine pattern in the cerebrospinal fluid from patients with GBS and CIDP
4. Interleukin-12
5. Common problems in clinical trials
6. Ouchterlony Ö Reverse ELISPOT assay for clinical analysis of cytokine production. I. Enumeration of gamma-interferon secreting cells
7. Comparison between ELISPOT methods for the detection of cytokine producing cells: Greater sensitivity and specificity using ELISA plates as compared to nitrocellulose membranes
8. Tumor necrosis factor-alpha and other cytokines in Guillain-Barré syndrome
9. IL-10 and IFN-γ in Guillain-Barré syndrome
10. Anti-GD1a antibody is associated with axonal but not demyelinating forms of Guillain-Barré syndrome