SCOPUS 정보 검색 플랫폼

Volumn 277, Issue 2, 2002, Pages 1107-1112

Decay-accelerating factor (DAF), complement receptor 1 (CR1), and factor H dissociate the complement AP C3 convertase (C3bBb) via sites on the type A domain of Bb

(5) Hourcade, Dennis E a,d Mitchell, Lynne b Kuttner Kondo, Lisa A c Atkinson, John P a Edward Medof, M b

a WASHINGTON UNIVERSITY SCHOOL OF MEDICINE (United States)

b CASE WESTERN RESERVE UNIVERSITY SCHOOL OF MEDICINE (United States)

c UNIVERSITY OF WASHINGTON (United States)

d Washington University (United States)

Author keywords

[No Author keywords available]

Indexed keywords

MUTATIONS;

DECAY (ORGANIC); POSITIVE IONS; PROTEINS;

BIOCHEMISTRY;

CLASSICAL COMPLEMENT PATHWAY C3 C5 CONVERTASE; COMPLEMENT COMPONENT C3B; COMPLEMENT FACTOR H; COMPLEMENT RECEPTOR; DECAY ACCELERATING FACTOR; PROTEIN SUBUNIT;

ALPHA HELIX; ARTICLE; BINDING AFFINITY; DISSOCIATION; MUTANT; MUTATION; PRIORITY JOURNAL; PROTEIN DOMAIN; PROTEIN PROTEIN INTERACTION; PROTEIN STABILITY;

ANTIGENS, CD55; CELL LINE; COMPLEMENT C3-C5 CONVERTASES; COMPLEMENT FACTOR H; CULTURE MEDIA, SERUM-FREE; HUMANS; MODELS, MOLECULAR; MULTIENZYME COMPLEXES; MUTAGENESIS, SITE-DIRECTED; PROTEIN STRUCTURE, TERTIARY; RECEPTORS, COMPLEMENT;

EID: 0037059776 PISSN: 00219258 EISSN: None Source Type: Journal
DOI: 10.1074/jbc.M109322200 Document Type: Article

Times cited : (55)

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