No association between a stop codon polymorphism in RAD52 and breast cancer risk

Cancer Epidemiology Biomarkers and Prevention

Volumn 11, Issue 10 I, 2002, Pages 1138-1139

  Han, Jiali ^a   Hankinson, Susan E ^a   De Vivo, Immaculata ^a   Colditz, Graham A ^a   Hunter, David J ^a  

^a HARVARD CENTER FOR CANCER PREVENTION   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

GENE PRODUCT; RAD52 PROTEIN; SERINE; UNCLASSIFIED DRUG;

ARTICLE; BREAST CANCER; CANCER RISK; CONTROLLED STUDY; DISEASE ASSOCIATION; FEMALE; GENETIC POLYMORPHISM; HUMAN; KNEE; LOGIC; MAJOR CLINICAL STUDY; POLYMENOPHOREA; PRIORITY JOURNAL; PROSPECTIVE STUDY; RISK ASSESSMENT; RISK FACTOR; STOP CODON;

BREAST NEOPLASMS; CASE-CONTROL STUDIES; CODON, TERMINATOR; DNA-BINDING PROTEINS; FEMALE; HUMANS; MUTATION, MISSENSE; ODDS RATIO; POLYMORPHISM, GENETIC; PROSPECTIVE STUDIES; RAD52 DNA REPAIR AND RECOMBINATION PROTEIN; RISK FACTORS;

EID: 0036802253     PISSN: 10559965     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (4)

1. Chen, J. J., Silver, D., Cantor, S., Livingston, D. M., and Scully, R. BRCA1, BRCA2, and Rad51 operate in a common DNA damage response pathway. Cancer Res., 59: 1752s-1756s, 1999.
2. Bell, D. W., Wahrer, D. C., Kang, D. H., MacMahon, M. S., FitzGerald, M. G., Ishioka, C., Isselbacher, K. J., Krainer, M., and Haber. D. A. Common nonsense mutations in RAD52. Cancer Res., 59: 3883-3888, 1999.
3. Haiman, C. A., Brown, M., Hankinson, S. E., Spiegelman, D., Colditz, G. A., Willett, W. C., Kantoff, P. W., and Hunter, D. J. The androgen receptor CAG repeat polymorphism and risk of breast cancer in the Nurses' Health Study. Cancer Res., 62: 1045-1049, 2002.
4. Shen. Z., Cloud, K. G., Chen, D. J., and Park, M. S. Specific interactions between the human RAD51 and RAD52 proteins. J. Biol. Chem., 271: 148-152, 1996.