-
2
-
-
0028784851
-
Protective effect of high density lipoprotein associated paraoxonase. Inhibition of the biological activity of minimally oxidised low density lipoprotein
-
Watson A.D., Berliner J.A., Hama S.Y., La Du B.N., Fault K.F., Fogelman A.M., Navab M. Protective effect of high density lipoprotein associated paraoxonase. Inhibition of the biological activity of minimally oxidised low density lipoprotein. J. Clin. Invest. 96:1995;2882-2891.
-
(1995)
J. Clin. Invest.
, vol.96
, pp. 2882-2891
-
-
Watson, A.D.1
Berliner, J.A.2
Hama, S.Y.3
La Du, B.N.4
Fault, K.F.5
Fogelman, A.M.6
Navab, M.7
-
3
-
-
0032537833
-
Mice lacking serum paraoxonase are susceptible to organophosphate toxicity and atherosclerosis
-
Shih D.M., Gu L., Xia Y.R., et al. Mice lacking serum paraoxonase are susceptible to organophosphate toxicity and atherosclerosis. Nature. 394:1998;284-287.
-
(1998)
Nature
, vol.394
, pp. 284-287
-
-
Shih, D.M.1
Gu, L.2
Xia, Y.R.3
-
4
-
-
0029937118
-
The human serum paraoxonase/arylesterase gene (PON1) is one member of a multigene family
-
Primo-Parmo S.L., Sorenson R.C., Teiber J., La Du B.N. The human serum paraoxonase/arylesterase gene (PON1) is one member of a multigene family. Genomics. 33:1996;498-507.
-
(1996)
Genomics
, vol.33
, pp. 498-507
-
-
Primo-Parmo, S.L.1
Sorenson, R.C.2
Teiber, J.3
La Du, B.N.4
-
5
-
-
0027486997
-
Molecular basis for the polymorphic forms of human serum paraoxonase/arylesterase: Glutamine or arginine at position 191, for the respective A or B allozymes
-
Adkins S., Gan K.N., Mody M., La Du B.N. Molecular basis for the polymorphic forms of human serum paraoxonase/arylesterase: Glutamine or arginine at position 191, for the respective A or B allozymes. Am. J. Hum. Genet. 52:1993;598-608.
-
(1993)
Am. J. Hum. Genet.
, vol.52
, pp. 598-608
-
-
Adkins, S.1
Gan, K.N.2
Mody, M.3
La Du, B.N.4
-
6
-
-
0027396833
-
The molecular basis of the human serum paraoxonase activity polymorphism
-
Humbert R., Adler D.A., Disteche C.M., Hassett C., Omiecinski C.J., Furlong C.E. The molecular basis of the human serum paraoxonase activity polymorphism. Nat. Genet. 3:1993;73-76.
-
(1993)
Nat. Genet.
, vol.3
, pp. 73-76
-
-
Humbert, R.1
Adler, D.A.2
Disteche, C.M.3
Hassett, C.4
Omiecinski, C.J.5
Furlong, C.E.6
-
7
-
-
0033973018
-
Promoter polymorphisms of human paraoxonase PON1 gene and serum paraoxonase activities and concentrations
-
Leviev I., James R.W. Promoter polymorphisms of human paraoxonase PON1 gene and serum paraoxonase activities and concentrations. Arterioscler. Thromb. Vasc. Biol. 20:2000;516-521.
-
(2000)
Arterioscler. Thromb. Vasc. Biol.
, vol.20
, pp. 516-521
-
-
Leviev, I.1
James, R.W.2
-
9
-
-
0032512704
-
Effect of the human serum paraoxonase 55 and 192 genetic polymorphisms on the protection by high density lipoprotein against low density lipoprotein oxidative modification
-
Mackness B., Mackness M.I., Arrol S., Turkie W., Durrington P.N. Effect of the human serum paraoxonase 55 and 192 genetic polymorphisms on the protection by high density lipoprotein against low density lipoprotein oxidative modification. FEBS Lett. 423:1998;57-60.
-
(1998)
FEBS Lett.
, vol.423
, pp. 57-60
-
-
Mackness, B.1
Mackness, M.I.2
Arrol, S.3
Turkie, W.4
Durrington, P.N.5
-
10
-
-
0032522985
-
Paraoxonase inhibits high-density lipoprotein oxidation and preserves its functions. A possible peroxidative role for paraoxonase
-
Aviram M., Rosenblat M., Bisgaier C.L., Newton R.S., Primo-Parno S.L., La Du B. Paraoxonase inhibits high-density lipoprotein oxidation and preserves its functions. A possible peroxidative role for paraoxonase. J. Clin. Invest. 101:1998;1581-1590.
-
(1998)
J. Clin. Invest.
, vol.101
, pp. 1581-1590
-
-
Aviram, M.1
Rosenblat, M.2
Bisgaier, C.L.3
Newton, R.S.4
Primo-Parno, S.L.5
La Du, B.6
-
11
-
-
0034738140
-
Human serum paraoxonases (PON1) Q and R selectively decrease lipid peroxides in human coronary and carotid atherosclerotic lesions: PON1 esterase and peroxidase-like activities
-
Aviram M., Hardak E., Vaya J., et al. Human serum paraoxonases (PON1) Q and R selectively decrease lipid peroxides in human coronary and carotid atherosclerotic lesions: PON1 esterase and peroxidase-like activities. Circulation. 101:2000;2510-2517.
-
(2000)
Circulation
, vol.101
, pp. 2510-2517
-
-
Aviram, M.1
Hardak, E.2
Vaya, J.3
-
12
-
-
0032945537
-
Paraoxonase protection of LDL against peroxidation is independent of its esterase activity towards paraoxon and is unaffected by the Q→R genetic polymorphism
-
Cao H., Girard-Globa A., Berthezene F., Moulin P. Paraoxonase protection of LDL against peroxidation is independent of its esterase activity towards paraoxon and is unaffected by the Q→R genetic polymorphism. J. Lipid Res. 40:1999;133-139.
-
(1999)
J. Lipid Res.
, vol.40
, pp. 133-139
-
-
Cao, H.1
Girard-Globa, A.2
Berthezene, F.3
Moulin, P.4
-
13
-
-
0028030547
-
Apo-lipoprotein profile in subjects with extracranial carotid atherosclerosis
-
Barbagallo C.M., Averna M.R., Novo S., et al. Apo-lipoprotein profile in subjects with extracranial carotid atherosclerosis. Int. Angiol. 13:1994;223-228.
-
(1994)
Int. Angiol.
, vol.13
, pp. 223-228
-
-
Barbagallo, C.M.1
Averna, M.R.2
Novo, S.3
-
14
-
-
0033135550
-
Relative importance of various risk factors for asymptomatic carotid atherosclerosis versus coronary heart disease incidence: The Atherosclerosis Risk in Communities Study
-
Sharrett A.R., Sorlie P.D., Chambless L.E., Folsom A.R., Hutchinson R.G., Heiss G., Szklo M. Relative importance of various risk factors for asymptomatic carotid atherosclerosis versus coronary heart disease incidence: The Atherosclerosis Risk in Communities Study. Am. J. Epidemiol. 149:1999;843-852.
-
(1999)
Am. J. Epidemiol.
, vol.149
, pp. 843-852
-
-
Sharrett, A.R.1
Sorlie, P.D.2
Chambless, L.E.3
Folsom, A.R.4
Hutchinson, R.G.5
Heiss, G.6
Szklo, M.7
-
15
-
-
8544255516
-
Awareness, treatment and control of hyperlipidaemia, hypertension and diabetes mellitus in a selected population of southern Italy
-
Gnasso A., Calindro M.C., Carallo C., et al. Awareness, treatment and control of hyperlipidaemia, hypertension and diabetes mellitus in a selected population of southern Italy. Eur. J. Epidemiol. 13:1997;421-428.
-
(1997)
Eur. J. Epidemiol.
, vol.13
, pp. 421-428
-
-
Gnasso, A.1
Calindro, M.C.2
Carallo, C.3
-
16
-
-
0000703842
-
The diagnosis of ischemic heart pain and intermittent claudication in field surveys
-
Rose G.A. The diagnosis of ischemic heart pain and intermittent claudication in field surveys. Bull. WHO. 27:1962;645-658.
-
(1962)
Bull. WHO
, vol.27
, pp. 645-658
-
-
Rose, G.A.1
-
17
-
-
0027291775
-
Human and rabbit paraoxonases: Purification, cloning, sequencing, mapping and role of polymorphism in organophosphate detoxification
-
Furlong C.E., Costa L.G., Hassett C., et al. Human and rabbit paraoxonases: Purification, cloning, sequencing, mapping and role of polymorphism in organophosphate detoxification. Chem. Biol. Interact. 87:1993;35-48.
-
(1993)
Chem. Biol. Interact.
, vol.87
, pp. 35-48
-
-
Furlong, C.E.1
Costa, L.G.2
Hassett, C.3
-
18
-
-
0020539832
-
Non-invasive evaluation of arteriosclerosis. Comparison of methods
-
Strandness D.E. Jr. Non-invasive evaluation of arteriosclerosis. Comparison of methods. Arteriosclerosis. 3:1983;103-116.
-
(1983)
Arteriosclerosis
, vol.3
, pp. 103-116
-
-
Strandness, D.E.1
-
19
-
-
0031004009
-
In vivo association between low wall shear stress and plaque in subjects with asymmetrical carotid atherosclerosis
-
Gnasso A., Irace C., Carallo C., De Franceschi M.S., Motti C., Mattioli P.L., Pujia A. In vivo association between low wall shear stress and plaque in subjects with asymmetrical carotid atherosclerosis. Stroke. 28:1997;993-998.
-
(1997)
Stroke
, vol.28
, pp. 993-998
-
-
Gnasso, A.1
Irace, C.2
Carallo, C.3
De Franceschi, M.S.4
Motti, C.5
Mattioli, P.L.6
Pujia, A.7
-
20
-
-
0032777923
-
Influence of the human paraoxonase polymorphism (PON1 192) on the carotid-wall thickening in a healthy population
-
Dessì M., Gnasso A., Motti C., et al. Influence of the human paraoxonase polymorphism (PON1 192) on the carotid-wall thickening in a healthy population. Coron. Artery Dis. 10:1999;595-599.
-
(1999)
Coron. Artery Dis.
, vol.10
, pp. 595-599
-
-
Dessì, M.1
Gnasso, A.2
Motti, C.3
-
21
-
-
0032102149
-
Lack of association between carotid intima-media thickness and paraoxonase gene polymorphism in non-insulin dependent diabetes mellitus
-
Cao H., Girard-Globa A., Serusclat A., et al. Lack of association between carotid intima-media thickness and paraoxonase gene polymorphism in non-insulin dependent diabetes mellitus. Atherosclerosis. 138:1998;361-366.
-
(1998)
Atherosclerosis
, vol.138
, pp. 361-366
-
-
Cao, H.1
Girard-Globa, A.2
Serusclat, A.3
-
22
-
-
0031690303
-
Paraoxonase PON1 polymorphism Leu-Met54 is associated with carotid atherosclerosis
-
Schmidt H., Schmidt R., Niederkorn K., et al. Paraoxonase PON1 polymorphism Leu-Met54 is associated with carotid atherosclerosis. Stroke. 29:1998;2043-2048.
-
(1998)
Stroke
, vol.29
, pp. 2043-2048
-
-
Schmidt, H.1
Schmidt, R.2
Niederkorn, K.3
-
23
-
-
0034020289
-
Paraoxonase gene polymorphisms are associated with carotid arterial wall thickness in subjects with familial hypercholesterolemia
-
Leus F.R., Wittekoek M.E., Prins J., Kastelein J.J., Voorbij H.A. Paraoxonase gene polymorphisms are associated with carotid arterial wall thickness in subjects with familial hypercholesterolemia. Atherosclerosis. 149:2000;371-377.
-
(2000)
Atherosclerosis
, vol.149
, pp. 371-377
-
-
Leus, F.R.1
Wittekoek, M.E.2
Prins, J.3
Kastelein, J.J.4
Voorbij, H.A.5
-
24
-
-
0032126007
-
Modulated serum activities and concentrations of paraoxonase in high density lipoprotein deficiency states
-
James R.W., Blatter Garin M.C., Calabresi L., et al. Modulated serum activities and concentrations of paraoxonase in high density lipoprotein deficiency states. Atherosclerosis. 139:1998;77-82.
-
(1998)
Atherosclerosis
, vol.139
, pp. 77-82
-
-
James, R.W.1
Blatter Garin, M.C.2
Calabresi, L.3
-
25
-
-
17344369707
-
The Gln-Arg192 polymorphism of human paraoxonase gene is not associated with coronary artery disease in Italian patients
-
Ombres D., Pannitteri G., Montali A., Candeloro A., Seccareccia F., Campagna F., Cantini R., Campa P.P., Ricci G., Arca M. The Gln-Arg192 polymorphism of human paraoxonase gene is not associated with coronary artery disease in Italian patients. Arterioscler. Thromb. Vasc. Biol. 18:1998;1611-1616.
-
(1998)
Arterioscler. Thromb. Vasc. Biol.
, vol.18
, pp. 1611-1616
-
-
Ombres, D.1
Pannitteri, G.2
Montali, A.3
Candeloro, A.4
Seccareccia, F.5
Campagna, F.6
Cantini, R.7
Campa, P.P.8
Ricci, G.9
Arca, M.10
-
26
-
-
0032974874
-
Paraoxonase 192 Gln/Arg gene polymorphism, coronary artery disease, and myocardial infarction in type 2 diabetes
-
Pfohl M., Koch M., Enderle M.D., Kuhn R., Fullhase J., Karsch K.R., Haring H.U. Paraoxonase 192 Gln/Arg gene polymorphism, coronary artery disease, and myocardial infarction in type 2 diabetes. Diabetes. 48:1999;623-627.
-
(1999)
Diabetes
, vol.48
, pp. 623-627
-
-
Pfohl, M.1
Koch, M.2
Enderle, M.D.3
Kuhn, R.4
Fullhase, J.5
Karsch, K.R.6
Haring, H.U.7
-
27
-
-
0029118601
-
Gln-Arg192 polymorphism of paraoxonase and coronary heart disease in type 2 diabetes
-
Ruiz J., Blanche H., James R.W., et al. Gln-Arg192 polymorphism of paraoxonase and coronary heart disease in type 2 diabetes. Lancet. 346:1995;869-872.
-
(1995)
Lancet
, vol.346
, pp. 869-872
-
-
Ruiz, J.1
Blanche, H.2
James, R.W.3
-
28
-
-
0033461179
-
Determination of paraoxonase (PON1) status requires more than genotyping
-
Richter R.J., Furlong C.E. Determination of paraoxonase (PON1) status requires more than genotyping. Pharmacogenetics. 9:1999;745-753.
-
(1999)
Pharmacogenetics
, vol.9
, pp. 745-753
-
-
Richter, R.J.1
Furlong, C.E.2
-
29
-
-
0035574446
-
Paraoxonase status in coronary heart disease: Are activity and concentration more important than genotype?
-
Mackness B., Davies G.K., Turkie W., et al. Paraoxonase status in coronary heart disease: Are activity and concentration more important than genotype? Arterioscler. Thromb. Vasc. Biol. 21:2001;1451-1457.
-
(2001)
Arterioscler. Thromb. Vasc. Biol.
, vol.21
, pp. 1451-1457
-
-
Mackness, B.1
Davies, G.K.2
Turkie, W.3
|