Altered Ca2+ homeostasis and impaired mitochondrial function in cardiomyopathy

Molecular and Cellular Biochemistry

Volumn 238, Issue 1-2, 2002, Pages 119-127

  Kuo, T H ^a   Zhu, L ^a   Golden, K ^a   Marsh, J D ^a   Bhattacharya, S K ^b   Liu, B F ^b  

^a WAYNE STATE UNIVERSITY SCHOOL OF MEDICINE   (United States)

^b UNIVERSITY OF TENNESSEE MEDICAL CENTER   (United States)

Author keywords

Apoptosis; Calcium homeostasis; Cardiomyopathy; Mitochondria; Necrosis; Sodium calcium exchange

Indexed keywords

CALCIUM CHANNEL BLOCKING AGENT; CALCIUM CHANNEL L TYPE; CALCIUM ION; DILTIAZEM; SODIUM CALCIUM EXCHANGE PROTEIN; SODIUM ION; CALCIUM; CASPASE; NICOTINAMIDE ADENINE DINUCLEOTIDE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CALCIUM CELL LEVEL; CALCIUM HOMEOSTASIS; CARDIOMYOPATHY; CELL CULTURE; CELL DEATH; CELL SURVIVAL; CLINICAL FEATURE; CONTROLLED STUDY; CORRELATION ANALYSIS; DISEASE ASSOCIATION; DISORDERS OF MITOCHONDRIAL FUNCTIONS; DRUG POTENCY; EXPERIMENTAL MODEL; HAMSTER; HEART MUSCLE FIBER MEMBRANE; INHIBITION KINETICS; MITOCHONDRIAL ENERGY TRANSFER; MUSCLE CELL; NONHUMAN; PATHOGENESIS; PROTEIN EXPRESSION; SODIUM CALCIUM EXCHANGE; TREATMENT OUTCOME; ANIMAL; ENZYME ACTIVATION; ENZYMOLOGY; HEART MITOCHONDRION; HEART MUSCLE; HOMEOSTASIS; ION EXCHANGE; METABOLISM; PATHOLOGY;

ANIMALIA; CRICETINAE;

ANIMALS; CALCIUM; CARDIOMYOPATHIES; CASPASES; CRICETINAE; ENZYME ACTIVATION; HOMEOSTASIS; ION EXCHANGE; MITOCHONDRIA, HEART; MYOCARDIUM; NAD; SODIUM-CALCIUM EXCHANGER;

EID: 0036715328     PISSN: 03008177     EISSN: None     Source Type: Journal    
DOI: 10.1023/A:1019967323419     Document Type: Article

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