Depressed PKA activity contributes to impaired SERCA function and is linked to the pathogenesis of glucose-induced cardiomyopathy

Journal of Molecular and Cellular Cardiology

Volumn 34, Issue 8, 2002, Pages 985-996

  Dutta, Kaushik ^a   Carmody, Marybeth W ^a   Cala, Steven E ^b   Davidoff, Amy J ^a  

^a UNIVERSITY OF NEW ENGLAND   (United States)

^b WAYNE STATE UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

Calcium transients; Diabetes; E C coupling; Hyperglycemia

Indexed keywords

ADENOSINE TRIPHOSPHATASE (CALCIUM); CYCLIC AMP DEPENDENT PROTEIN KINASE; N [2 (4 BROMOCINNAMYLAMINO)ETHYL] 5 ISOQUINOLINESULFONAMIDE; PROTEIN SERCA; UNCLASSIFIED DRUG;

ANIMAL CELL; ANIMAL MODEL; ARTICLE; BETA ADRENERGIC STIMULATION; CALCIUM TRANSPORT; CARDIOMYOPATHY; CELL CULTURE; CELL KINETICS; CONTROLLED STUDY; CULTURE MEDIUM; ENDOPLASMIC RETICULUM; ENZYME ACTIVITY; ENZYME INHIBITION; INHIBITION KINETICS; MALE; MUSCLE CELL; NONHUMAN; PRIORITY JOURNAL; PROTEIN CONTENT; PROTEIN PHOSPHORYLATION; RAT; REGULATORY MECHANISM; SARCOPLASMIC RETICULUM; TRANSPORT KINETICS;

EID: 0036702693     PISSN: 00222828     EISSN: None     Source Type: Journal    
DOI: 10.1006/jmcc.2002.2035     Document Type: Article

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