Ca2+-induced changes in SNAREs and synaptotagmin I correlate with triggered exocytosis from chromaffin cells: Insights gleaned into the signal transduction using trypsin and botulinum toxins

Journal of Cell Science

Volumn 115, Issue 13, 2002, Pages 2791-2800

  Lawrence, Gary W ^a   Dolly, J Oliver ^a  

^a Department of Biological Science   (United Kingdom)

Author keywords

Clostridial neurotoxins; Large dense core granules; Secretion; SNAP 25; Synaptobrevin

Indexed keywords

BOTULINUM TOXIN; CALCIUM BINDING PROTEIN; CALCIUM ION; CALCIUM SENSING RECEPTOR; CATECHOLAMINE; PHOSPHOLIPID; SNARE PROTEIN; SYNAPTOBREVIN; SYNAPTOSOMAL ASSOCIATED PROTEIN 25; SYNAPTOTAGMIN; SYNTAXIN; TRYPSIN; ADENOSINE TRIPHOSPHATE; BOTULINUM TOXIN B; CALCIUM; DIAGNOSTIC AGENT; MEMBRANE PROTEIN; NERVE PROTEIN; PROTEINASE K; SYNAPTOTAGMIN I; VESICULAR TRANSPORT PROTEIN;

ANIMAL CELL; ARTICLE; CALCIUM BINDING; CALCIUM CELL LEVEL; CALCIUM SIGNALING; CARBOXY TERMINAL SEQUENCE; CATTLE; CELL MEMBRANE PERMEABILITY; CHROMAFFIN CELL; CONTROLLED STUDY; CORRELATION ANALYSIS; DATA ANALYSIS; EXOCYTOSIS; IN VITRO STUDY; NONHUMAN; POLYACRYLAMIDE GEL ELECTROPHORESIS; PRIORITY JOURNAL; PROTEIN ANALYSIS; PROTEIN DEGRADATION; PROTEIN PROTEIN INTERACTION; PROTEIN SECRETION; PROTEIN STRUCTURE; SENSITIVITY ANALYSIS; SIGNAL TRANSDUCTION; ADRENAL MEDULLA; ANIMAL; CELL CULTURE; CELL MEMBRANE; DOSE RESPONSE; DRUG EFFECT; METABOLISM; PHYSIOLOGY; SECRETION; SECRETORY VESICLE;

ANIMALIA; BOS TAURUS;

ADENOSINE TRIPHOSPHATE; ADRENAL MEDULLA; ANIMALS; BOTULINUM TOXINS; CALCIUM; CALCIUM SIGNALING; CALCIUM-BINDING PROTEINS; CATECHOLAMINES; CATTLE; CELL MEMBRANE; CELLS, CULTURED; CHROMAFFIN CELLS; DOSE-RESPONSE RELATIONSHIP, DRUG; ENDOPEPTIDASE K; EXOCYTOSIS; MEMBRANE GLYCOPROTEINS; MEMBRANE PROTEINS; NERVE TISSUE PROTEINS; QA-SNARE PROTEINS; R-SNARE PROTEINS; SECRETORY VESICLES; SIGNAL TRANSDUCTION; SNARE PROTEINS; SYNAPTOSOMAL-ASSOCIATED PROTEIN 25; SYNAPTOTAGMIN I; SYNAPTOTAGMINS; TRYPSIN; VESICULAR TRANSPORT PROTEINS;

EID: 0036629350     PISSN: 00219533     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

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