Human congenital long QT syndrome: More than previously thought?

Trends in Pharmacological Sciences

Volumn 23, Issue 6, 2002, Pages 249-251

  Attali, Bernard ^a  

^a TEL AVIV UNIVERSITY   (Israel)

Author keywords

[No Author keywords available]

Indexed keywords

ALDOSTERONE; ANGIOTENSIN; CORTICOTROPIN; POTASSIUM CHANNEL; POTASSIUM ION; PROTEIN; PROTEIN KCNE1; PROTEIN KCNQ1; RENIN; SODIUM ION; UNCLASSIFIED DRUG; KCNQ1 PROTEIN, HUMAN; POTASSIUM CHANNEL KCNQ; POTASSIUM CHANNEL KCNQ1; POTASSIUM CHANNEL PROTEIN I(SK); VOLTAGE GATED POTASSIUM CHANNEL;

ALPHA CHAIN; BETA CHAIN; CARDIOVASCULAR RISK; CELL MEMBRANE TRANSPORT; EPITHELIUM; GENE MUTATION; HORMONE ACTION; HUMAN; HYPERALDOSTERONISM; HYPOKALEMIA; INNER EAR; KIDNEY; KNOCKOUT MOUSE; LONG QT SYNDROME; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN FUNCTION; PROTEIN LOCALIZATION; SHORT SURVEY; ANIMAL; ARTICLE; GENETICS; MUTATION;

ANIMALS; HUMANS; HYPERALDOSTERONISM; HYPOKALEMIA; KCNQ POTASSIUM CHANNELS; KCNQ1 POTASSIUM CHANNEL; LONG QT SYNDROME; MUTATION; POTASSIUM CHANNELS; POTASSIUM CHANNELS, VOLTAGE-GATED;

EID: 0036591655     PISSN: 01656147     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0165-6147(02)02029-1     Document Type: Review

References (20)

1. KvLQT1 and IsK (minK) proteins associate to form the IKs cardiac potassium current
2. Coassembly of KvLQT1 and minK (IsK) proteins to form cardiac IKs potassium channel
3. Voltage-gated ion channels and hereditary disease
4. Molecular and genetic basis of electrophysiological disease: The long QT syndrome
5. KCNQ potassium channels: Physiology, pathophysiology, and pharmacology
6. Altered potassium balance and aldosterone secretion in a mouse model of human congenital long QT syndrome
7. + channel in heart rate control of repolarization in a murine engineered model of Jervell and Lange-Nielsen syndrome
8. The surdo-cardiac syndrome
9. + fluxes in mouse proximal tubule
10. Differential expression of KvLQT1 and its regulator IsK in mouse epithelia
11. + channel and a mutant causing Liddle syndrome: A quantitative approach
12. + channel) is expressed in glomerulosa cells of rat adrenal cortex and inhibited by angiotensin II
13. + channel KCNQ1 is essential for gastric acid secretion
14. Mice disrupted for the KvLQT1 potassium channel regulator IsK gene accumulate mature T cells
15. Targeted disruption of the Kvlqt1 gene causes deafness and gastric hyperplasia in mice
16. A superfamily of small potassium channel subunits: Form and function of the MinK-related peptides (MiRPs)
17. MIRP2 forms potassium channels in skeletal muscle with Kv3.4 and is associated with periodic paralysis
18. Mink-related peptide 1, a β subunit for the HCN ion channel subunit family enhances expression and speeds activation
19. Mink-related peptide 1 associates with Kv4.2 and modulates its gating function